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Correction

Published online by Cambridge University Press:  02 January 2018

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Abstract

Type
Correction
Copyright
Copyright © iStockphoto/Devon Stephens 

Cookson J (2010) Seeing the evidence: learning from images in neuro-science. .

The author would like to make the following post-publication clarifications to his article, published in March issue of Advances.

On p. 83, the caption to Fig. 1 should read:

Kandel's model for the cellular and molecular basis of declarative memory formation in the hippocampus. The image combines information gained from his studies of giant marine snail neurons and studies of vertebrate hippocampus. Artist: Philip Wilson.

On p. 84, the fourth paragraph in the left-hand column should end:

The cellular mechanisms involved in long-term potentiation have also been implicated in hypotheses about the pathophysiology of depression. This was first discussed in detail by Reid & Stewart (2001; see also Zaman & Zaman 2001). The image illustrates a role for dopamine in signalling salience for memory. In the snail, serotonin plays a role similar to that postulated for dopamine in Fig. 1, potentiating glutamate synapses. Harmer et al (2009) argue that serotonin plays a part in determining the affective salience of cues, which has relevance for depression. The role of CPEB may cast light on the pathogenesis of prion diseases.

The reference list on p. 84 should include:

Harmer CJ, Goodwin GM, Cowen PJ (2009) Why do antidepressants take so long to work? A cognitive neuropsychological model of antidepressant drug action. British Journal of Psychiatry 195: 102–8.

Reid IC, Stewart CA (2001) How antidepressants work. New perspectives on the pathophysiology of depressive disorder. British Journal of Psychiatry 178: 299–303.

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