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    This article has been cited by the following publications. This list is generated based on data provided by CrossRef.

    Dick, Danielle M. Adkins, Amy E. and Kuo, Sally I-Chun 2016. Genetic influences on adolescent behavior. Neuroscience & Biobehavioral Reviews,


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    Marshall, E. J. 2014. Adolescent Alcohol Use: Risks and Consequences. Alcohol and Alcoholism, Vol. 49, Issue. 2, p. 160.


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    Palmer, R. H. C. Young, S. E. Corley, R. P. Hopfer, C. J. Stallings, M. C. and Hewitt, J. K. 2013. Stability and Change of Genetic and Environmental Effects on the Common Liability to Alcohol, Tobacco, and Cannabis DSM-IV Dependence Symptoms. Behavior Genetics, Vol. 43, Issue. 5, p. 374.


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    Light, Laney S. McCoy, Thomas P. Thompson, Martie P. Spitler, Hugh D. Sutfin, Erin L. and Rhodes, Scott D. 2011. Modeling the Rutgers Alcohol Problem Index (RAPI): A comparison of statistical methods. Addiction Research & Theory, Vol. 19, Issue. 6, p. 510.


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Personality at Ages 16 and 17 and Drinking Problems at Ages 18 and 25: Genetic Analyses of Data from FinnTwin 16–25

  • Richard J. Viken (a1), Jaakko Kaprio (a2) and Richard J. Rose (a3)
  • DOI: http://dx.doi.org/10.1375/twin.10.1.25
  • Published online: 01 February 2012
Abstract
Abstract

We enrolled more than 3500 same-sex twins from 5 consecutive Finnish birth cohorts into a longitudinal study as each cohort reached age 16. Twins completed the Psychopathic Deviate (Pd) Scale of the Minnesota Multiphasic Personality Inventory at baseline, Sensation Seeking Scale items as each cohort reached age 17, and later, at average ages 18.5 and 25, the Rutgers Alcohol Problem Index (RAPI). Using raw maximum likelihood estimation, we fit a Cholesky model to the 4 variables assessed at 4 ages across the 4 twin types; we estimated genetic and environmental influences on the stability of alcohol problems across development and the genetic and environmental contributions to predictive correlations between adolescent personality and later alcohol-related behavior problems. With one exception, the phenotypic, genetic, and environmental correlations were very similar for males and females. The exception was that the lagged associations of Pd and RAPI reflect a higher genetic correlation among males than females and a higher environmental correlation among females than males. Our analyses suggest that developmental changes underlying variation in alcohol problems from late adolescence to early adulthood differ for males and females. In males, the main change is decreased variation due to shared environmental effects; the magnitude of genetic effects is stable over time, and the high genetic correlation, .95, suggests that the same genetic influences are important at both ages. Among females, in contrast, genetic influences decline in magnitude from age 18 to 25, and at least part of the genetic effect evident at age 25 differs from the genetic effect evident at age 18.

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Corresponding author
*Address for correspondence: Richard J. Rose, Department of Psychological and Brain Sciences, Indiana University, 1011 East 10th Street, Bloomington, IN 47405, USA.
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Twin Research and Human Genetics
  • ISSN: 1832-4274
  • EISSN: 1839-2628
  • URL: /core/journals/twin-research-and-human-genetics
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