Hostname: page-component-848d4c4894-4hhp2 Total loading time: 0 Render date: 2024-06-03T07:59:38.201Z Has data issue: false hasContentIssue false
  • English
  • Français

A Non-Permanent Tonic Pupil in Rheumatoid Arteritis

Published online by Cambridge University Press:  18 September 2015

David I. Victor ,
W. Richard Green ,
Walter J. Stark  and
Frank B. Walsh
David I. Victor
Affiliation:
Eye Pathology Laboratory, Wilmer Institute and Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, Maryland
W. Richard Green*
Affiliation:
Eye Pathology Laboratory, Wilmer Institute and Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, Maryland
Walter J. Stark
Affiliation:
Eye Pathology Laboratory, Wilmer Institute and Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, Maryland
Frank B. Walsh
Affiliation:
Eye Pathology Laboratory, Wilmer Institute and Department of Pathology, The Johns Hopkins Medical Institutions, Baltimore, Maryland
*
Eye Pathology Laboratory, The Johns Hopkins Hospital, 601 N. Broadway, BaltimoreMaryland 21205
Rights & Permissions [Opens in a new window]

Summary:

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

A 76-year-old male with a severely deforming rheumatoid arthritis, eosinophilic!, polymyositis, and episcleritis developed a transient tonic pupil. The episcleritis, and a muscle biopsy revealing an occlusive arteritis with eosinophilia, suggest that a widespread rheumatoid arteritis caused a reversible ischemic insult to the ciliary ganglion and thus created a transient denervation of the pupil.

Type
Research Article
Information
Canadian Journal of Neurological Sciences , Volume 4 , Issue 3 , August 1977 , pp. 209 - 212
Copyright
Copyright © Canadian Neurological Sciences Federation 1977

References

REFERENCES

Davis, R.H., Daroff, R.B. and Hoyt, W.F. (1968). Tonic pupil after temporal arteritis. Lancet 1: 822.CrossRefGoogle ScholarPubMed
Farooki, Z.Q., Brough, A.J. and Green, E.W. (1974). Necrotizing arteritis. Am. J. Dis. Child 128: 837–840.Google ScholarPubMed
Littlewood, J.M. and Lewis, G.M. (1963). The Holmes-Adie syndrome in a boy with acute juvenile rheumatism and bilateral syndactyly. Arch. Dis. Child 38: 86–88.CrossRefGoogle Scholar
Lowenstein, O. and Loewenfeld, I.E. (1965). Pupillotonic pseudotabes. Survey Ophthalmol. 10: 129–185.Google ScholarPubMed
Lyne, A.J. and Pitkeathley, D.A. (1968). Episcleritis and scleritis. Arch. Ophthalmol. 80: 171–176.CrossRefGoogle ScholarPubMed
Panush, R.D., Franco, A.E. and Schur, P.H. (1971). Rheumatoid arthritis associated with eosinophilia. Ann. Intern.Med. 75; 199–205.CrossRefGoogle ScholarPubMed
Ramos, M. and Mandybur, T.I. (1975). Cerebral vasculitis in rheumatoid arthritis. Arch. Neurol. 32: 271–275.CrossRefGoogle ScholarPubMed
Schmid, F.R., Cooper, N.S., Ziff, M. and McEwen, C. (1961). Arteritis in rheumatoid arthritis. Am. J. Med. 30: 56–83.CrossRefGoogle ScholarPubMed
Steinburg, V.L. and Parry, C.B. (1961). Electromyographic changes in rheumatoid arthritis. Br. Med. J. 5226: 630–632.CrossRefGoogle Scholar
Zeek, P.M. (1953). Periarteritis nodosa and other forms of necrotizing angitis. New Eng. J. Med. 248: 764–772.CrossRefGoogle Scholar