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Neuropsychiatry of corticosubcortical relations Aphasia and depression as paradigms

Published online by Cambridge University Press:  18 September 2015

W. van den Bergh
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Summary

In an introduction the evolution of a neuropsychological and neuropsychiatrie approach in the two last decades is discussed. The role of the basal ganglia, the thalamus and their cortical connections in aphasia is discussed. This review is given as a paradigm to demonstrate a neuropsychiatrie approach of psychiatric disorders. The role of corticosubcortical relations in depression is discussed.

Keywords

neuropsychiatrybasal gangliathalamuscorticosubcortical relationsaphasiadepressionneuropsychiatriebasale gangliathalamuscorticosubcorticale relatiesafasiedepressie
Type
Research Article
Information
Acta Neuropsychiatrica , Volume 2 , Issue 3 , September 1990 , pp. 61 - 65
Copyright
Copyright © Scandinavian College of Neuropsychopharmacology 1990

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References

Literatuur

1.Changeux, JP. L'homme neuronal. Paris: Fayard 1983; 335–6.Google Scholar
2.De Ajuriaguerra, J, Hécaen, H. Le cortex cérébral: étude neuropsycho-pathologique. Paris: Masson, 1964.Google Scholar
3.Luria, AR. Higher cortical functions in man. New York, Basic Books, 1966.Google Scholar
4.Kinsbourne, M, red. Cerebral hemisphere function in depression. Washington, American Psychiatric Press, 1988.Google Scholar
5.Andreasen, NC, red. Can schizophrenia be localized in the brain? Washington, American Psychiatric Press, 1986.Google Scholar
6.Baxter, LR. Brain imaging as a tool in establishing a theory of brain pathology in obsessive compulsive disorder. J clin Psychiat 1990; 51(2,Suppl):22–5.Google ScholarPubMed
7Flor-Henry, P.Hysteria. In: Frederiks, JAM, red. Handbook of clinical neurology, Vol. 2 (46). Amsterdam: Elsevier, 1985; 573–83.Google Scholar
8.Alexander, MP, Naeser, MA, Palumbo, CL. Correlations of subcortical CT lesion sites and aphasia profiles. Brain 1987; 110:961–91.CrossRefGoogle ScholarPubMed
9.Metter, EJ, Wasterlain, CG, Kuhl, DEet al.18-FDG PET tomography in a study on aphasia. Ann Neurol 1981; 10:173–83.CrossRefGoogle Scholar
10.Perani, D, Vallar, G, Cappa, Set al.Aphasia and neglect after subcortical stroke: a clinical/cerebral perfusion correlation study. Brain 1987;110:1211–29.CrossRefGoogle ScholarPubMed
11.Cambier, J, Graveleau, P. Thalamic syndromes. In: Frederiks, JAM, red. Handbook of Clinical Neurology, Vol. 1 (45). Amsterdam: Elsevier, 1985:87–98.Google Scholar
12.Luria, AR. Sub 3, 237–41.Google Scholar
13.McFarling, D, Rothi, LJ, Heilman, KM. Transcortical aphasia from ischaemic infarcts of the thalamus. A report of two cases. J Neurol Neurosurg, Psychiat 1982; 45:107–12.CrossRefGoogle ScholarPubMed
14.Penfield, W, Roberts, L. Speech and brain mechanisms. Princeton: Princeton University Press, 1959:138–234.Google Scholar
15.Crosson, B. Subcortical functions in language: a working model. Brain Lang 1985; 25:257–92.CrossRefGoogle Scholar
16.Kuhl, DE, Phelps, ME, Kowell, APet al.Effects of stroke on local cerebral metabolism and perfusion: mapping by emission computed tomography of 18-FDG and 13-NH3. Ann Neurol 1980;8:47–60.CrossRefGoogle Scholar
17.Robinson, RG, Kubos, KL, Starr, LBet al.Mood disorders in stroke patients: importance of location of lesion. Brain 1984; 107: 81–93.CrossRefGoogle ScholarPubMed
18.Phelps, ME, Mazziotta, JC, Gerner, RHet al.Human cerebral glucose metabolism in affective disorders: drug-free states and pharmacological effects. J cereb Blood Flow Metab 1983; 3(51): 57–8.Google Scholar
19.Baxter, LR, Phelps, ME, Mazziotta, JCet al.Cerebral metabolic rates for glucose in mood disorders: studies with positron emission tomography and fluorodeoxyglucose F18. Arch gen Psychiat 1985; 42: 441–7.CrossRefGoogle Scholar
20.Baxter, LR, Schwartz, JM, Phelps, MEet al.Reduction of prefrontal cortex glucose metabolism common to three types of depression. Arch Gen Psychiat 1989; 46:243–9.CrossRefGoogle ScholarPubMed
21.Devous, MD, Rush, AJ, Schlesser, MAet al.Single-photon tomographic determination of rCBF in psychiatric disorders. J Nucl Med 1984; 25:P57.Google Scholar
22.Schaffer, CE, Davidson, RJ, Saron, C. Frontal and parietal EEG asymmetries in depressed and non-depressed subjects. Biol Psychiat 1983;18:753–62.Google Scholar
23.Schatzberg, AF, Elliott, GR, Lerlinger, JEet al.Topographic mapping in depressed patients. In: Duffy, FH, red. Topographic mapping of brain electrical activity. Boston: Butterworths, 1986:389–92.CrossRefGoogle Scholar
24Flor-Henry, P, Fromm, D, Schopflocher, D. Neuropsychological test performance in depressed patients before and after drug therapy. Biol Psychiat 1984; 19:55–72.Google Scholar
25.Taylor, MA, Redfield, J, Abrams, R. Neuropsychological dysfunction in schizophrenia and affective disease. Biol Psychiat 1981; 16:467–78.Google ScholarPubMed
26.Kronfol, Z, Hamsher, KD, Nigre, Ket al.Depression and hemispheric functions: changes associated with unilateral ECT. Br J Psychiat 1978; 132:560–7.CrossRefGoogle ScholarPubMed
27.Kinsbourne, M. Hemispheric interactions in depression. In: Kinsbourne M, red. Sub 4, 135–162.Google Scholar
28.Willner, P. Depression: apsychobiological synthesis. New York: Wiley, 1985:85–100.Google Scholar
29.Starkstein, SE, Robinson, RG, Price, TR. Comparison of cortical and subcortical lesions in the production of poststroke mood disorders. Brain 1987; 110:1045–59.CrossRefGoogle ScholarPubMed
30.Starkstein, SE, Robinson, RG, Berthier, MLet al.Differential mood changes following basal ganglia vs thalamic lesions. Arch Neurol 1988; 45:725–30.CrossRefGoogle ScholarPubMed
31.Starkstein, SE, Cohen, BS, Fedoroff, Pet al.Relationship between anxiety disorders and depressive disorders in patients with cerebrovascular injury. Arch gen Psychiat 1990; 47:246–51.CrossRefGoogle ScholarPubMed
32.Starkstein, SE, Preziosi, TJ, Bolduc, PL, Robinson, RG. Depression in Parkinson's disease. J nervmentdis 1990; 178:27–31.Google ScholarPubMed
33.Starkstein, SE, Preziosi, TJ, Berthier, MLet al.Depression and cognitive impairments in Parkinson's disease. Brain 1989; 112:1141–53.CrossRefGoogle ScholarPubMed
34.Fibiger, HC. The neurobiological substrates of depression in Parkinson's disease: a hypothesis. Can J neurolsci 1984; 11: 105–7.CrossRefGoogle ScholarPubMed
35.Javoy-Agid, F, Agid, Y. Is the mesocortical dopaminergic system involved in Parkinson's disease? Neurol 1980; 30:1326–30.CrossRefGoogle Scholar
36.Mendez, MF, Adams, NL, Lewandowski, KS. Neurobehavioral changes associated with caudate lesions. Neurol 1989; 39:349–54.CrossRefGoogle ScholarPubMed
37.Krishnan, KRR, Figiel, GS. Neurobehavioral changes with caudate lesions: letterto the editor. Neurol 1989; 39:1410–1.CrossRefGoogle Scholar
38.Alexander, GE, DeLong, MR, Strick, PL. Parallel organization of functionally segregated circuits linking basal ganglia and cortex. Ann Rev Neurosci 1986;9:357–81.CrossRefGoogle ScholarPubMed
39.Buchsbaum, MS, DeLisi, LE, Holcomb, HHet al.Antero-posterior gradient in Cerebral glucose use in schizophrenia and affective disorders. Arch Gen Psychiat 1984; 41:1159–66.CrossRefGoogle Scholar
40.Willner, P., sub 28.Google Scholar