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Jha, Mithilesh Kumar Lee, In-Kyu and Suk, Kyoungho 2016. Metabolic reprogramming by the pyruvate dehydrogenase kinase–lactic acid axis: Linking metabolism and diverse neuropathophysiologies. Neuroscience & Biobehavioral Reviews, Vol. 68, p. 1.
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Costa, Filipa Alexandra Leite and Moreira Neto, Fani Lourença 2015. Células gliales satélite de ganglios sensoriales: su papel en el dolor. Brazilian Journal of Anesthesiology (Edicion en Espanol), Vol. 65, Issue. 1, p. 73.
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Hanani, Menachem 2015. Role of satellite glial cells in gastrointestinal pain. Frontiers in Cellular Neuroscience, Vol. 9,
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Zhao, Ya-Juan Liu, Yang Zhao, Yin-Hua Li, Qiang Zhang, Min and Chen, Yong-Jin 2015. Activation of satellite glial cells in the trigeminal ganglion contributes to masseter mechanical allodynia induced by restraint stress in rats. Neuroscience Letters, Vol. 602, p. 150.
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Retamal, Mauricio A. Alcayaga, Julio Verdugo, Christian A. Bultynck, Geert Leybaert, Luc SÃ¡ez, Pablo J. FernÃ¡ndez, Ricardo LeÃ³n, Luis E. and SÃ¡ez, Juan C. 2014. Opening of pannexin- and connexin-based channels increases the excitability of nodose ganglion sensory neurons. Frontiers in Cellular Neuroscience, Vol. 8,
Satellite glial cells (SGCs) undergo phenotypic changes and divide the following injury into a peripheral nerve. Nerve injury, also elicits an immune response and several antigen-presenting cells are found in close proximity to SGCs. Silencing SCG-specific molecules involved in intercellular transport (Connexin 43) or glutamate recycling (glutamine synthase) can dramatically alter nociceptive responses of normal and nerve-injured rats. Transducing SGCs with glutamic acid decarboxylase can produce analgesia in models of trigeminal pain. Taken together these data suggest that SGCs may play a role in the genesis or maintenance of pain and open a range of new possibilities for curing neuropathic pain.
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