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10 - Exhibiting Gabriel Lippmann: A Collaborative Challenge
- Edited by Hanin Hannouch
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- Book:
- Gabriel Lippmann's Colour Photography
- Published by:
- Amsterdam University Press
- Published online:
- 16 November 2022
- Print publication:
- 13 June 2022, pp 251-272
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Abstract
This essay aims to show how the nature of cultural goods initiates specific collaborative approaches. Photo Elysée, Museum for Photography owns 138 Lippmann interferential plates, one of the largest collections in the world. Donated by descendants of Lippmann's in-laws in the 1990s, these plates are among the wonders of the collection, which counts over one million photographic objects. This article aims to explain what such a collection brings to the institution in terms of opening up to external disciplines and collaborations which, in turn, nourish the museum's work in a much broader way. At the intersection of the history of colour photography, science and contemporary art, Lippmann plates push the museum beyond its borders.
Keywords: Gabriel Lippmann, museum collections, museology, contemporary physics, exhibitions, preservation
With the largest collection of original interferential photochromes by Gabriel Lippmann in the world, Photo Elysée in Lausanne (Switzerland) has recently launched an ambitious research programme to deepen knowledge not only of Lippmann process, in which other researchers and institutions are also interested, but also of Lippmann's role in the history of photography. The complexity of interferential photochromy, Gabriel Lippmann's special position between the scientific, photographic and holographic worlds, and the diffusion and circulation of the process in Europe and throughout the world make it necessary to cross-reference fields of expertise to gain a better understanding of this subject. For this reason, Photo Elysée has embarked on an important interdisciplinary collaboration, which will open up new perspectives of collaborations for the institution for future projects.
An Atypical Collection
In 1985, when Pierre-Yves Suter learned that the Musée de l’Elysée (former name of Photo Elysée), a few kilometres from his home, was dedicated to preserving photography in all its forms, he persuaded his father, Gabriel Suter, to bequeath the Lippmann interferential photochromes he owned to this new institution. Indeed, Gabriel Suter and his sister, Antoinette Monition Suter, were the heirs of the physicist Gabriel Lippmann (1845–1921). Although they had never known their illustrious great-uncle, their mother, Charlotte Cherbuliez, had often spoken of him. She and her sister were very close to the childless Lippmann couple (Suter 1999).
Lymphatic absorption of α-linolenic acid in rats fed flaxseed oil-based emulsion
- Leslie Couëdelo, Carole Boué-Vaysse, Laurence Fonseca, Emeline Montesinos, Sandrine Djoukitch, Nicole Combe, Maud Cansell
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- Journal:
- British Journal of Nutrition / Volume 105 / Issue 7 / 14 April 2011
- Published online by Cambridge University Press:
- 17 November 2010, pp. 1026-1035
- Print publication:
- 14 April 2011
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The bioavailability of α-linolenic acid (ALA) from flaxseed oil in an emulsified form v. a non-emulsified form was investigated by using two complementary approaches: the first one dealt with the characterisation of the flaxseed oil emulsion in in vitro gastrointestinal-like conditions; the second one compared the intestinal absorption of ALA in rats fed the two forms of the oil. The in vitro study on emulsified flaxseed oil showed that decreasing the pH from 7·3 to 1·5 at the physiological temperature (37°C) induced instantaneous oil globule coalescence. Some phase separation was observed under acidic conditions that vanished after further neutralisation. The lecithin used to stabilise the emulsions inhibited TAG hydrolysis by pancreatic lipase. In contrast, lipid solubilisation by bile salts (after lipase and phospholipase hydrolysis) was favoured by preliminary oil emulsification. The in vivo absorption of ALA in thoracic lymph duct-cannulated rats fed flaxseed oil, emulsified or non-emulsified, was quantified. Oil emulsification significantly favoured the rate and extent of ALA recovery as measured by the maximum ALA concentration in the lymph (Cmax = 14 mg/ml at 3 h in the emulsion group v. 9 mg/ml at 5 h in the oil group; P < 0·05). Likewise, the area under the curve of the kinetics was significantly higher in the emulsion group (48 mg × h/ml for rats fed emulsion v. 26 mg × h/ml for rats fed oil; P < 0·05). On the whole, ALA bioavailability was improved with flaxseed oil ingested in an emulsified state. Data obtained from the in vitro studies helped to partly interpret the physiological results.
12 - Genetics of human susceptibility to infection and hepatic disease caused by schistosomes
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- By Alain J. Dessein, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Sandrine Marquet, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Carole Eboumbou Moukoko, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Hèlia Dessein, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Laurent Argiro, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Sandrine Henri, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Dominique Hillaire, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Christophe Chevillard, Immunologie et Génétique des Maladies Parasitaires, Institut National de la Santé et de la Recherche Médicale, Laboratoire de Parasitologie-Mycologie, Faculté de Médecine, Marseille, France, Nasureldin El Wali, Institute of Nuclear Medicine and Molecular Biology, University of Gezira, Wad Medani, Sudan, Mubarak Magzoub, Institute of Nuclear Medicine and Molecular Biology, University of Gezira, Wad Medani, Sudan, Laurent Abel, Génétique Humaine des Maladies Infectieuses, Institut National de la Santé et de la Recherche Médicale, Faculté de Médecine Necker, Paris, Virmondes Rodrigues, Jr, Faculty of Medicine do Triangulo Mineiro, Ubéraba, Brazil, Aluizio Prata, Faculty of Medicine do Triangulo Mineiro, Ubéraba, Brazil, Gachuhi Kimani, Kenya Medical Research Institute, Biomedical Sciences Research Centre, Nairobi, Kenya
- Edited by Richard Bellamy, Kintampo Health Research Centre, Ghana
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- Book:
- Susceptibility to Infectious Diseases
- Published online:
- 14 August 2009
- Print publication:
- 22 December 2003, pp 337-360
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Schistosome infections cause much suffering in millions of people living in tropical regions of Africa, Asia, and South America (Prata, 1987; Chitsulo et al., 2000). The most severe clinical symptoms affect the kidneys and urinary tract. However, schistosomes also cause various other disorders such as heart failure and neurological diseases. Three species of schistosome are responsible for most human infections (Schistosoma mansoni, Schistosoma japonicum, and Schistosoma haematobium). These species are found in different geographical locations, have different vectors, and cause different symptoms. Schistosomes are multicellular parasites that are disseminated as free swimming larvae (cercariae) in ponds, lakes, and rivers by snails. Humans become infected when they stay in contaminated water for a few minutes. The cercariae penetrate the human skin and develop into male or female adult schistosomes within 5 or 6 weeks. These small worms (Fig. 12.1A) can live in the vascular system of their vertebrate host for 2 to 5 years. Schistosomes do not multiply within their vertebrate host. The female worms, however, lay hundreds of eggs per day in the mesenteric or vesical veins of their host. Most of the symptoms associated with these infections are caused by the inflammation that is induced by the immunogenic and toxic substances produced by the eggs. The chronic cellular reaction that develops around the eggs is organised in a granuloma (Von Lichtenberg, 1962; Warren et al., 1967).