The dissipation of ion-acoustic surface waves propagating in a semi-bounded and collisional plasma which has a boundary with vacuum is theoretically investigated and this result is used for the analysis of edge-relevant plasma simulated by Divertor Plasma Simulator-2 (DiPS-2). The collisional damping of the surface wave is investigated for weakly ionized plasmas by comparing the collisionless Landau damping with the collisional damping as follows: (1) the ratio of ion temperature $({T_i})$ to electron temperature $({T_e})$ should be very small for the weak collisionality $({T_i}/{T_e} \ll 1)$; (2) the effect of collisionless Landau damping is dominant for the small parallel wavenumber, and the decay constant is given as $\gamma \approx{-} \sqrt {\mathrm{\pi }/2} {k_\parallel }{\lambda _{De}}\omega _{pi}^2/{\omega _{pe}}$; and (3) the collisional damping dominates for the large parallel wavenumber, and the decay constant is given as $\gamma \approx{-} {\nu _{in}}/16$, where ${\nu _{in}}$ is the ion–neutral collisional frequency. An experimental simulation of the above theoretical prediction has been done in the argon plasma of DiPS-2, which has the following parameters: plasma density ${n_e} = (\textrm{2--9)} \times \textrm{1}{\textrm{0}^{11}}\;\textrm{c}{\textrm{m}^{ - 3}}$, ${T_e} = 3.7- 3.8\;\textrm{eV}$, ${T_i} = 0.2- 0.3\;\textrm{eV}$ and collision frequency ${\nu _{in}} = 23- 127\;\textrm{kHz}$. Although the wavelength should be specified with the given parameters of DiPS-2, the collisional damping is found to be $\gamma = ( - 0.9\;\textrm{to}\; - 5) \times {10^4}\;\textrm{rad}\;{\textrm{s}^{ - 1}}$ for ${k_\parallel }{\lambda _{De}} = 10$, while the Landau damping is found to be $\gamma = ( - 4\;\textrm{to}\; - 9) \times {10^4}\;\textrm{rad}\;{\textrm{s}^{ - 1}}$ for ${k_\parallel }{\lambda _{De}} = 0.1$.

The purpose of the present study was to characterize the Ca2+ channels in smooth muscle cells from human stomach and to examine the effects of osmotic swelling on the channel activity. Ca2+ channel current with either Ca2+ or Ba2+ as charge carrier was recorded from freshly isolated smooth muscle cells using the conventional whole-cell patch clamp technique. The degree of cell swelling as a result of hypotonic challenge was monitored using a video image analysis system. The changes in intracellular Ca2+ concentration ([Ca2+]i) were measured by microfluorimetry. The pharmacological and voltage activation profile suggests a typical dihydropyridine-sensitive L-type Ca2+ current. Cell swelling, induced by hypotonic challenge, enhanced the amplitude of currents through L-type Ca2+ channels without significant effects on steady-state voltage dependency. After treatment with the L-type Ca2+ channel agonist Bay K 8644 (0.1-2 µM), no further significant increase in calcium channel current or corresponding [Ca2+]i transients were provoked by the swelling. The above results demonstrated that the presence of L-type Ca2+ current in smooth muscle cells of the human stomach and the augmentation of the current are closely associated with the volume increase resulting from hypotonic swelling. Experimental Physiology (2000) 85.5, 497-504.