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Cereal Food Poisoning and its Relation to the Etiology of Pellagra

Published online by Cambridge University Press:  15 May 2009

Ralph Stockman
Affiliation:
Professor of Materia Medica and Therapeutics, University of Glasgow
J. M. Johnston
Affiliation:
Scottish Department of Health, lately Lecturer on Pharmacology, University of Glasgow.
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Pellagra is a comparatively modern disease. The first account of it dates from 1735 when under the name mal de la rosa it was very fully described by Gaspar Casal who at that time practised at Oviedo and found it endemic among the Asturian peasantry. In Lombardy it was recognised about 1730 and only much later (1771) was systematically described by Frapolli as pellagra (rough skin), but Strambio, who was physician to a special hospital for pellagrins at Legnano, states (1784) that many of his patients told him that their fathers and grandfathers had suffered from the disease. Early in the nineteenth century it was comparatively common in Gascony and about 1836 it was beginning to be recognised in Roumania. In the United States sporadic cases seem to have been noticed in 1864, and since 1907 it has increased to an extent which has seriously disquieted the public health authorities. In Egypt, Turkey, Mexico, the Balkans, Brazil, the West Indies and other countries it is common and well known. Wherever it has appeared endemically it has followed on the introduction of maize as a field-crop and its adoption (by the rural population especially) as a chief and staple article of diet.

Type
Research Article
Copyright
Copyright © Cambridge University Press 1933

References

page 205 note 1 J. Amer. Med. Assoc. (1920), p. 21.Google Scholar

page 205 note 2 Étiologie pellagreuse, Borladu, 1882.Google Scholar

page 206 note 1 Arch. Int. Med. 25, 450.Google Scholar

page 210 note 1 J. Chem. Soc. 66, II, 286.Google Scholar

page 210 note 2 J. Biol Chem. 54, 375.Google Scholar

page 211 note 1 The Diseases of China, 2nd ed.Google Scholar

page 211 note 2 Japan J. of Dermat, and Urolog. (1929), 29, 65.Google Scholar

page 211 note 3 Brit. Med. J. (1912), ii, 1773; (1908), ii, 1608.Google Scholar

page 212 note 1 Trans. Soc. Trap. Med. (19131914), 7, 47Google Scholar; Trans. Roy. Soc. Med. (19291930), 23, 740.Google Scholar

page 212 note 2 J. Trap. Med. and Hyg. (1918), 21, 153; (1920), 23, 46.Google Scholar

page 213 note 1 Watson, , Seventh Annual Report of the Board of Control, for 1921, and Eleventh Annual Report, for 1924.Google Scholar

page 213 note 2 Brown, and Low, , Edin. Med. J. (1909), 3, 197.Google Scholar

page 214 note 1 Sambon, and Chalmers, , Brit. Med. J. (1912), ii, 1093.CrossRefGoogle Scholar

page 214 note 2 Henderson, , Rev. of Neurol. (1916), 13, 579.Google Scholar

page 214 note 3 Heusinger, (1856) Studien über den Ergotismus, Marburg. Thieme (1930), Veröffentl. a. d. Geb. d. Medicinalverwaltung, 32, 5.Google Scholar

page 214 note 4 Stockman, (1917), Edin. Med. J. (Lathyrus peas)Google Scholar; (1931), J. Hygiene, 37 (various peas)Google Scholar; Anderson, , Howard, and Simonsen, (19241925), Ind. J. Med. Res. 12 (Vicia angustifolia).Google Scholar

page 214 note 5 See Thieme, loc. cit.Google Scholar

page 216 note 1 J. Biol. Chem. (1916), 25, 239.Google Scholar

page 217 note 1 Brain (1931), 54, 347.Google Scholar