Hostname: page-component-848d4c4894-x24gv Total loading time: 0 Render date: 2024-05-20T03:35:07.428Z Has data issue: false hasContentIssue false
  • English
  • Français

Some epithelial changes in fluorosis

Published online by Cambridge University Press:  15 May 2009

Leo Spira
Rights & Permissions [Opens in a new window]

Extract

Core share and HTML view are not available for this content. However, as you have access to this content, a full PDF is available via the ‘Save PDF’ action button.

Like the skin and its appendages, the teeth, nails and hair, so also other tissues of ectodermal origin are here shown to be affected by fluorine. The action of fluorine consists in lowering the level of the calcium in the blood and tissues. The epithelia of the mouth and the salivary glands, of the nasopharynx, the conjunctivae and the lachrymal glands may be affected. Signs and symptoms are thus produced which are closely similar to those usually attributed primarily to infection. It is suggested that an infection accompanying chronic fluorine poisoning is of a secondary nature, and that it is brought about by the loss of calcium in the body.

Type
Research Article
Information
Journal of Hygiene , Volume 43 , Issue 6 , September 1944 , pp. 400 - 401
Copyright
Copyright © Cambridge University Press 1944

References

REFERENCES

Ainsworth, N. J. (1933). Brit. Dent. J. 55, 233.Google Scholar
Broekema, J. H. (1933). Acta derm.-venereol., Stockh., 14, 113.Google Scholar
Dean, H. T. (1936). J. Amer. Med. Ass. 107, 1269.CrossRefGoogle Scholar
Dean, H. T. & Elvove, E. (1936). Amer. J. Publ. Hlth, 26, 567.CrossRefGoogle Scholar
Erdheim, J. (1906). Mitt. Grenzgeb. Med. Chir. 16, 632.Google Scholar
Friedmann, M. (1921). Arch. Derm. Syph., Berlin, 135, 161.Google Scholar
Gaud, Charnot & Langlais, (1935). Odontologie, 73, 188.Google Scholar
Gilford, H. (1904). Practitioner, 73, 188.Google Scholar
Goeckermann, W. H. (1920). Arch. Derm. Syph., Chicago, 1, 396.CrossRefGoogle Scholar
Hill, A. M. (1933). Arch. Derm. Syph., Chicago, 28, 66.CrossRefGoogle Scholar
Jacobsen, A. W. (1928). J. Amer. Med. Ass. 90, 686.Google Scholar
Josephson, E. M. & Lerner, C. (1934). Arch. Derm. Syph., Chicago, 29, 703.CrossRefGoogle Scholar
Kaznelson, P., Reimann, F. & Weiner, W. (1929). Klin. Wschr. 81, 1071.CrossRefGoogle Scholar
Lachmann, A. (1941). Acta Med. Scand., Suppl. 121.Google Scholar
McEwen, E. L. (1916). J. Cutan. Dis. 34, 15.Google Scholar
MacKee, G. M. & Andrews, G. C. (1924). Arch. Derm. Syph., Chicago, 10, 673.CrossRefGoogle Scholar
Mendes da Costa, S. & Van der Valk, J. W. (1919). Urol. Cutan. Rev. 23, 159.Google Scholar
Nicolle, G. & Halipré, A. (1895). Ann. Derm. Syph., Paris, 6, 675, 804.Google Scholar
Spira, L. (1928). Franco-Brit. Med. Rev. 5, 1, 61.Google Scholar
Spira, L. (1933). The Clinical Aspect of Chronic Poisoning by Aluminium and its Alloys. London.Google Scholar
Spira, L. (1942 a). Edinb. Med. J. 49, 707.Google Scholar
Spira, L. (1942 b). J. Hyg., Camb., 42, 500.Google Scholar
Spira, L. (1943 a). J. Hyg., Camb., 43, 69.Google Scholar
Spira, L. (1943 b). To be published.Google Scholar
Spira, L. (1943 c). Edinb. Med. J. 50, 237.Google Scholar
Thurnam, J. (1848). Proc. Roy. Med. Chir. Soc. 31, 71.CrossRefGoogle Scholar
Weech, A. A. (1929). Amer. J. Dis. Child. 37, 766.CrossRefGoogle Scholar
Wenner, W. F. (1931). J. Lab. Clin. Med. 16, 341.Google Scholar