Hjort, Jonas Sølvsten, Mikkel and Wüst, Miriam 2017. Universal Investment in Infants and Long-Run Health: Evidence from Denmark’s 1937 Home Visiting Program. American Economic Journal: Applied Economics, Vol. 9, Issue. 4, p. 78.
Minton, Jon Shaw, Richard Green, Mark A Vanderbloemen, Laura Popham, Frank and McCartney, Gerry 2017. Visualising and quantifying ‘excess deaths’ in Scotland compared with the rest of the UK and the rest of Western Europe. Journal of Epidemiology and Community Health, Vol. 71, Issue. 5, p. 461.
Jones, Alex L. 2017. The influence of shape and colour cue classes on facial health perception. Evolution and Human Behavior,
Ogasawara, Kota 2017. Persistence of pandemic influenza on the development of children: Evidence from industrializing Japan. Social Science & Medicine, Vol. 181, p. 43.
Acquah, Joseph Kofi Dahal, Roshani and Sloan, Frank A. 2017. 1918 Influenza Pandemic: In Utero Exposure in the United States and Long-Term Impact on Hospitalizations. American Journal of Public Health, Vol. 107, Issue. 9, p. 1477.
Hayward, Adam D. Rigby, Francesca L. and Lummaa, Virpi 2016. Early-life disease exposure and associations with adult survival, cause of death, and reproductive success in preindustrial humans. Proceedings of the National Academy of Sciences, Vol. 113, Issue. 32, p. 8951.
Li, Li Boland, Mary Regina Miotto, Riccardo Tatonetti, Nicholas P. and Dudley, Joel T. 2016. Replicating Cardiovascular Condition-Birth Month Associations. Scientific Reports, Vol. 6, Issue. 1,
Zhang, Qi Deng, Yafei Lai, Wenjing Guan, Xiao Sun, Xiongshan Han, Qi Wang, Fangjie Pan, Xiaodong Ji, Yan Luo, Hongqin Huang, Pei Tang, Yuan Gu, Liangqi Dan, Guorong Yu, Jianhua Namaka, Michael Zhang, Jianxiang Deng, Youcai and Li, Xiaohui 2016. Maternal inflammation activated ROS-p38 MAPK predisposes offspring to heart damages caused by isoproterenol via augmenting ROS generation. Scientific Reports, Vol. 6, Issue. 1,
Boudreaux, Michel H. Golberstein, Ezra and McAlpine, Donna D. 2016. The long-term impacts of Medicaid exposure in early childhood: Evidence from the program's origin. Journal of Health Economics, Vol. 45, p. 161.
Crimmins, Eileen M. 2015. Physiological Differences Across Populations Reflecting Early Life and Later Life Nutritional Status and Later Life Risk for Chronic Disease. Journal of Population Ageing, Vol. 8, Issue. 1-2, p. 51.
Vaiserman, A. M. 2015. Epigenetic programming by early-life stress: Evidence from human populations. Developmental Dynamics, Vol. 244, Issue. 3, p. 254.
Boland, Mary Regina Shahn, Zachary Madigan, David Hripcsak, George and Tatonetti, Nicholas P 2015. Birth month affects lifetime disease risk: a phenome-wide method. Journal of the American Medical Informatics Association, Vol. 22, Issue. 5, p. 1042.
Harper, Sam 2015. Invited Commentary: A-P-C … It's Easy as 1-2-3!. American Journal of Epidemiology, Vol. 182, Issue. 4, p. 313.
Doblhammer, Gabriele and Fritze, Thomas 2015. Month of Birth and Dementia Late in Life. KZfSS Kölner Zeitschrift für Soziologie und Sozialpsychologie, Vol. 67, Issue. S1, p. 217.
Parman, John 2015. Childhood health and sibling outcomes: Nurture Reinforcing nature during the 1918 influenza pandemic. Explorations in Economic History, Vol. 58, p. 22.
Ho, Jessica Y. 2015. Early-Life Environmental Exposures and Height, Hypertension, and Cardiovascular Risk Factors Among Older Adults in India. Biodemography and Social Biology, Vol. 61, Issue. 2, p. 121.
Cocoros, Noelle M. Lash, Timothy L. Ozonoff, Al Nørgaard, Mette DeMaria, Alfred Andreasen, Viggo and Sørensen, Henrik Toft 2014. Prenatal influenza exposure and cardiovascular events in adulthood. Influenza and Other Respiratory Viruses, Vol. 8, Issue. 1, p. 83.
Prenatal exposure to the 1918 influenza pandemic (Influenza A, H1N1 subtype) is associated with ⩾20% excess cardiovascular disease at 60 to 82 years of age, relative to cohorts born without exposure to the influenza epidemic, either prenatally or postnatally (defined by the quarter of birth), in the 1982–1996 National Health Interview Surveys of the USA. Males showed stronger effects of influenza on increased later heart disease than females. Adult height at World War II enlistment was lower for the 1919 birth cohort than for those born in adjacent years, suggesting growth retardation. Calculations on the prevalence of maternal infections indicate that prenatal exposure to even uncomplicated maternal influenza may have lasting consequences later in life. These findings suggest novel roles for maternal infections in the fetal programming of cardiovascular risk factors that are independent of maternal malnutrition.
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