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Third mobile window associated with suspected otosclerotic foci in two patients with an air–bone gap

Published online by Cambridge University Press:  23 August 2010

V Van Rompaey ,
J Potvin ,
L van den Hauwe  and
P Van de Heyning
V Van Rompaey*
Affiliation:
Department of Otorhinolaryngology and Head and Neck Surgery, Antwerp University Hospital, Edegem, Belgium
J Potvin
Affiliation:
Department of Otorhinolaryngology and Head and Neck Surgery, Antwerp University Hospital, Edegem, Belgium
L van den Hauwe
Affiliation:
Department of Radiology, Antwerp University Hospital, Edegem, Belgium
P Van de Heyning
Affiliation:
Department of Otorhinolaryngology and Head and Neck Surgery, Antwerp University Hospital, Edegem, Belgium
*
Address for correspondence: Dr Vincent Van Rompaey, Department of Otorhinolaryngology and Head and Neck Surgery, University Hospital of Antwerp, Wilrijkstraat 10, 2650 Edegem, Belgium Fax: +32 3 825 44 51 E-mail: vincent.vanrompaey@ua.ac.be
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Abstract

Objective:

To demonstrate the need for computed tomography imaging of the temporal bone in patients clinically suspected of otosclerosis who present with atypical symptoms or audiological findings.

Case reports:

We present two patients with bilateral conductive hearing loss and suspected otosclerosis in whom third mobile window lesions were revealed. The first patient had bilateral large vestibular aqueducts and bilateral fenestral otosclerotic foci. Computed tomography imaging of the second case revealed bilateral superior semicircular canal dehiscence and bilateral cochlear clefts, mimicking an otosclerotic focus in the fissula ante fenestram.

Conclusion:

Differentiating third mobile window lesions from otosclerosis as the cause of a conductive hearing loss is essential before considering stapes surgery, as such treatment would be unnecessary and potentially harmful.

Keywords

OtosclerosisVestibular AqueductConductive Hearing LossComputed TomographyX-Ray
Type
Clinical Records
Information
The Journal of Laryngology & Otology , Volume 125 , Issue 1 , January 2011 , pp. 89 - 92
Copyright
Copyright © JLO (1984) Limited 2010

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References

1 Mudry, A. Adam Politzer (1835–1920) and the description of otosclerosis. Otol Neurotol 2006;27:276–81CrossRefGoogle Scholar
2 Merchant, SN, Nakajima, HH, Halpin, C, Nadol, JB Jr, Lee, DJ, Innis, WP et al. Clinical investigation and mechanism of air-bone gaps in large vestibular aqueduct syndrome. Ann Otol Rhinol Laryngol 2007;116:532–41CrossRefGoogle ScholarPubMed
3 Merchant, SN, Rosowski, JJ. Conductive hearing loss caused by third-window lesions of the inner ear. Otol Neurotol 2008;29:282–9CrossRefGoogle ScholarPubMed
4 Minor, LB. Clinical manifestations of superior semicircular canal dehiscence. Laryngoscope 2005;115:1717–27CrossRefGoogle ScholarPubMed
5 Emmett, JR. The large vestibular aqueduct syndrome. Am J Otol 1985;6:387–415Google ScholarPubMed
6 Govaerts, PJ, Casselman, J, Daemers, K, De Ceulaer, G, Somers, T, Offeciers, FE. Audiological findings in large vestibular aqueduct syndrome. Int J Pediatr Otorhinolaryngol 1999;51:157–64CrossRefGoogle ScholarPubMed
7 Roditi, RE, Eppsteiner, RW, Sauter, TB, Lee, DJ. Cervical vestibular evoked myogenic potentials (cVEMPs) in patients with superior canal dehiscence syndrome (SCDS). Otolaryngol Head Neck Surg 2009;141:24–8CrossRefGoogle ScholarPubMed
8 Ribeiro, TK, Fayad, JN. A large vestibular aqueduct syndrome associated with otosclerosis. Otol Neurotol 2008;29:725–6CrossRefGoogle ScholarPubMed
9 Mikulec, AA, McKenna, MJ, Ramsey, MJ, Rosowski, JJ, Herrmann, BS, Rauch, SD et al. Superior semicircular canal dehiscence presenting as conductive hearing loss without vertigo. Otol Neurotol 2004;25:121–9CrossRefGoogle ScholarPubMed
10 Chadwell, JB, Halsted, MJ, Choo, DI, Greinwald, JH, Benton, C. The cochlear cleft. AJNR Am J Neuroradiol 2004;25:21–4Google ScholarPubMed