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B.02 Recessive mutations in ATP8A2 cause severe hypotonia, cognitive impairment, hyperkinetic movement disorders and progressive optic atrophy

Published online by Cambridge University Press:  27 June 2018

HJ McMillan
Affiliation:
(Ottawa)
A Telegrafi
Affiliation:
(Gaithersburg)
A Singleton
Affiliation:
(Gaithersburg)
M Cho
Affiliation:
(Gaithersburg)
D Lelli
Affiliation:
(Ottawa)
FC Lynn
Affiliation:
(Vancouver)
J Griffin
Affiliation:
(Louisville)
A Asamoah
Affiliation:
(Louisville)
T Rinne
Affiliation:
(Nijmegen)
CE Erasmus
Affiliation:
(Nijmegen)
DA Koolen
Affiliation:
(Nijmegen)
CA Haaxma
Affiliation:
(Nijmegen)
B Keren
Affiliation:
(Paris)
D Doummar
Affiliation:
(Paris)
C Mignot
Affiliation:
(Paris)
I Thompson
Affiliation:
(Toronto)
L Velsher
Affiliation:
(Toronto)
M Dehghani
Affiliation:
(Yazd)
M Vahidi Mehrjardi
Affiliation:
(Yazd)
R Maroofian
Affiliation:
(London)
M Tchan
Affiliation:
(Sydney)
C Simons
Affiliation:
(St. Lucia)
J Christodoulou
Affiliation:
(Melbourne)
E Martín-Hernández
Affiliation:
(Madrid)
MJ Guillen Sacoto
Affiliation:
(Gaithersburg)
LB Henderson
Affiliation:
(Gaithersburg)
H McLaughlin
Affiliation:
(Gaithersburg)
LL Molday
Affiliation:
(Vancouver)
RS Molday
Affiliation:
(Vancouver)
G Yoon
Affiliation:
(Toronto)
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Abstract

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Background:ATP8A2 mutations have only recently been associated with human disease. We present the clinical features from the largest cohort of patients with this disorder reported to date. Methods: An observational study of 9 unreported and 2 previously reported patients with biallelic ATP8A2 mutations was carried out at multiple centres. Results: The mean age of the cohort was 9.4 years old (range: 2.5-28 yrs). All patients demonstrated developmental delay, severe hypotonia and movement disorders: chorea/choreoathetosis (100%), dystonia (27%) or facial dyskinesia (18%). Hypotonia was apparent at birth (70%) or before 6 months old (100%). Optic atrophy was observed in 75% of patients who had a funduscopic examination. MRI of the brain was normal for most patients with a small proportion showing mild cortical atrophy (30%), delayed myelination (20%) and/or hypoplastic optic nerves (20%). Epilepsy was seen in two older patients. Conclusions:ATP8A2 gene mutations have emerged as a cause of a novel phenotype characterized by developmental delay, severe hypotonia and hyperkinetic movement disorders. Optic atrophy is common and may only become apparent in the first few years of life, necessitating repeat ophthalmologic evaluation. Early recognition of the cardinal features of this condition will facilitate diagnosis of this disorder.

Type
PLATFORM PRESENTATIONS
Copyright
© The Canadian Journal of Neurological Sciences Inc. 2018