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Cognitive diffusion of susceptibles driven by new infection cases in SIS epidemic models: frequency-dependent incidence

Published online by Cambridge University Press:  10 June 2026

Guodong Liu
Affiliation:
School of Mathematical Sciences, Shanghai Jiao Tong University, China
Hao Wang*
Affiliation:
Department of Mathematical and Statistical Sciences, University of Alberta , Canada
Xiaoyan Zhang
Affiliation:
School of Mathematics, Shandong University, China
*
Corresponding author: Hao Wang; Email: hao8@ualberta.ca
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Abstract

In this study, we develop epidemic reaction-diffusion models by incorporating the dependency of the diffusion rate of susceptible individuals on new infection cases, employing both Fickian and Fokker–Planck-type diffusion laws. As the first part of a two-part series, we focus on epidemics driven by frequency-dependent incidence. We explore linear, exponential and algebraic relationships between diffusion rate of the susceptible population and new infection cases to provide deeper biological insights. Our analysis establishes the global existence of solutions and characterizes the threshold dynamics using basic reproduction numbers. We find that in quasilinear parabolic systems, the Fokker–Planck-type diffusion law tends to induce spatial segregation of susceptible and infected individuals, while the Fickian law favours spatial homogenization of susceptible individuals. Additionally, the Fokker–Planck-type model, where the diffusion rate of infected individuals depends on new recovery cases, more accurately captures the cognitive diffusion behaviour of individuals.

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Papers
Creative Commons
Creative Common License - CCCreative Common License - BY
This is an Open Access article, distributed under the terms of the Creative Commons Attribution licence (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted re-use, distribution and reproduction, provided the original article is properly cited.
Copyright
© The Author(s), 2026. Published by Cambridge University Press
Figure 0

Figure 1. The threshold dynamics are given by (a)–(c) for (1.5) and (d)–(f) for (1.6). (a) and (d): $\beta =0.1$ and $\gamma =0.3$; (b) and (e): $\beta =\gamma =0.1$; (c) and (f): $\beta =0.3$ and $\gamma =0.1$.

Figure 1

Figure 2. The infection-recovery rates. (a) $\beta =6\cos x+6.6$, $\gamma =\cos x+1.5$; (b) $\beta =6\cos x+6.6$, $\gamma =\sin x+1.5$; (c) $\beta =5e^{-160(x-0.2)^2}+4$, $\gamma =5e^{-160(x-0.8)^2}+2$.

Figure 2

Figure 3. The distribution of steady states for $k=1$, $\eta =0.0001$, $\beta =6\cos x+6.6$, $\gamma =\cos x+1.5$.

Figure 3

Table 1. Segregation indices and infection fraction of $k=1$, $\eta =0.0001$, $\beta =6\cos x+6.6$, $\gamma =\cos x+1.5$

Figure 4

Table 2. Segregation indices and infection fraction of $k=1$, $\eta =0.0001$, $\beta =6\cos x+6.6$, $\gamma =\sin x+1.5$

Figure 5

Figure 4. The distribution of steady states for $k=1$, $\eta =0.0001$, $\beta =6\cos x+6.6$, $\gamma =\sin x+1.5$.

Figure 6

Figure 5. The distribution of steady states for $k=1$, $\eta =0.0001$, $\beta =5e^{-160(x-0.2)^2}+4$, $\gamma =5e^{-160(x-0.8)^2}+2$.

Figure 7

Table 3. Segregation indices and infection fraction of $k=1$, $\eta =0.0001$, $\beta =5e^{-160(x-0.2)^2}+4$, $\gamma =5e^{-160(x-0.8)^2}+2$

Figure 8

Figure 6. The distribution of steady states for $k=0.1$, $\eta =1$, $\beta =6\cos x+6.6$, $\gamma =\cos x+1.5$.

Figure 9

Table 4. Segregation indices and infection fraction of $k=0.1$, $\eta =1$, $\beta =6\cos x+6.6$, $\gamma =\cos x+1.5$