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The influence of social network on depressive and anxiety symptoms during the COVID-19 pandemic: findings from a Swedish cohort study

Published online by Cambridge University Press:  23 December 2025

Gillian L. Murphy*
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden
Emily E. Joyce
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden
Anikó Lovik
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden Institute of Psychology, Leiden University, The Netherlands
Elísabet U. Gísladóttir
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden
Katalin Vincze
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden Department of Medical Epidemiology and Biostatistics, Karolinska Institute, Stockholm, Sweden
Anna K. Kähler
Affiliation:
Department of Medical Epidemiology and Biostatistics, Karolinska Institute, Stockholm, Sweden
Emma M. Frans
Affiliation:
Department of Medical Epidemiology and Biostatistics, Karolinska Institute, Stockholm, Sweden
Unnur A. Valdimarsdóttir
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden Centre of Public Health Sciences, Faculty of Medicine, School of Health Sciences, University of Iceland, Reykjavik, Iceland Department of Epidemiology, Harvard TH Chan School of Public Health, Boston, Massachusetts, USA
Patrick F. Sullivan
Affiliation:
Department of Medical Epidemiology and Biostatistics, Karolinska Institute, Stockholm, Sweden Departments of Genetics and Psychiatry, University of North Carolina, Chapel Hill, North Caroline, USA
Mary Barker
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden
Fang Fang
Affiliation:
Institute of Environmental Medicine, Karolinska Institute, Stockholm, Sweden
*
Correspondence: Gillian L. Murphy. Email: glmurphy30@gmail.com.
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Abstract

Background

The COVID-19 pandemic disrupted daily social interactions, potentially affecting mental health. Understanding the risk of depressive and anxiety symptoms is essential for guiding mental health strategies during future crises.

Aims

To explore how social networks influenced mental health outcomes during the pandemic and how these relationships changed over time.

Method

Data from the Omtanke2020 study, a prospective cohort study of Swedish adults, were analysed using structural equation modelling (N = 10 918). Surveys at baseline and follow-up at 6 and 12 months assessed social networks, including structural components (e.g. relationship status, frequency of social contact) and perceived components (e.g. emotional support from family, feeling safe at home). Cross-lagged panel modelling was used to observe changes over time in the associations between social network indicators and depressive and anxiety symptoms.

Results

Stronger perceived social support – specifically closeness to family, perceived warmth or love from others and increased societal cohesion – were negatively correlated with depressive and anxiety symptoms across all time points (β coefficients = −0.14 to −0.23, all P < 0.001). Social network variables consistently predicted mental health outcomes, with effect sizes remaining relatively stable over time (β coefficient = −0.17 at baseline, β coefficient = −0.21 at 1-year follow-up).

Conclusions

This study highlights the protective role of the social network – namely perceived social support – in combatting depressive and anxiety symptoms during the COVID-19 pandemic. Interventions that strengthen close interpersonal ties and community cohesion may help mitigate mental health impacts during future public health crises.

Information

Type
Paper
Creative Commons
Creative Common License - CCCreative Common License - BY
This is an Open Access article, distributed under the terms of the Creative Commons Attribution licence (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted re-use, distribution and reproduction, provided the original article is properly cited.
Copyright
© The Author(s), 2025. Published by Cambridge University Press on behalf of Royal College of Psychiatrists
Figure 0

Fig. 1 Flowchart of the study sample.

Figure 1

Table 1 Baseline characteristics of the study sample

Figure 2

Fig. 2 Path diagrams of adjusted cross-sectional models at baseline and 6- and 12-month follow-ups. Anx, anxiety symptoms; Dep, depressive symptoms; MH, mental health; SN, social network; SS4, family closeness; SS5, perceived warmth or love from others; SS6, increased societal cohesion. Dashed lines indicate paths from latent variables to the first observed endogenous variables, which are set to a normalisation constraint of 1.00; solid lines indicate other regression paths between latent variables SN and MH, as well as those between latent and observed variables. Two-headed curved arrows indicate the standardised variance for each observed variable. All paths presented in the diagram are statistically significant. The fully adjusted baseline model was used, adjusting for age, gender, previous psychiatric diagnosis, physical comorbidities, body mass index, smoking status, CAGE Substance Abuse Screening Tool score, physical activity, recruitment type, previous COVID-19 infection, COVID-19-related worries, economic difficulties, employment status and response period. Models for the 6- and 12-month follow-ups were adjusted for previous COVID-19 infection, COVID-19-related worries, economic difficulties, employment status and response period.

Figure 3

Table 2 Model fit statistics for all structural equation models after trimming procedure applied

Figure 4

Fig. 3 Path diagram of the fully adjusted cross-lagged panel model at baseline (0) and 1-year follow-up (12). Anx, anxiety symptoms; Dep, depressive symptoms; MH, mental health; SN, social network; SS4, family closeness; SS5, perceived warmth or love from others; SS6, increased societal cohesion. Dashed lines indicate paths from latent variables to the first observed endogenous variables, which are set to a normalisation constraint of 1.00; solid lines indicate other regression paths between latent variables SN and MH, as well as those between latent and observed variables. Two-headed curved arrows indicate the standardised variance for each observed variable. All paths presented in the diagram are significant. The fully adjusted model (model 3) was implemented using the following baseline variables: age, gender, previous psychiatric diagnosis, physical comorbidities, body mass index, smoking status, CAGE Substance Abuse Screening Tool score, physical activity, recruitment type, previous COVID-19 infection, COVID-19-related worries, economic difficulties, employment status and response period. The 1-year model is adjusted for previous COVID-19 infection, COVID-19-related worries, economic difficulties, employment status and response period.

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