Published online by Cambridge University Press: 27 July 2009
INTRODUCTION: A VIEW OF THE ETIOLOGY OF PTSD
Posttraumatic stress disorder is the better researched consequence of traumatic events. In its current formulation (APA, 1994) the disorder is essentially linked to the triggering event: It cannot be diagnosed in the absence of a traumatic event, and its core symptoms of reexperiencing and avoidance must refer to the traumatic event.
This perspective implies a causal link between the event and subsequent PTSD. Such a link is also intuitively appealing and frequently appears in survivors' narratives. For an external observer as well, the association between a traumatic event and PTSD appears to have some truth to it because many survivors' lives are dramatically changed by an encounter with extreme adversity or evil. Thus, from both the survivor's perspective and that of involved observers, the traumatic event is the cause of PTSD.
From a scientific point of view, however, this is only half true because many trauma survivors do not develop PTSD. A traumatic event, accordingly, is a necessary, but certainly not a sufficient cause of PTSD. If so, then what are the alternatives?
The oldest alternative to simple causation refers to individual differences in vulnerability. Historically, this mainly concerned predisposing factors, an attribution that often carried value judgments. The latter could extend from assuming a personality defect in those who broke down under stress to more understandingly relegating the causality to the wear and tear of the central nervous system by adverse living circumstances (e.g., McEwen, 2000) or birth circumstances (e.g., Meaney et al., 1991; see also Bagot et al., this volume).
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