In 1994 and 1995, several new reports appeared regarding cellular mechanisms of LTD. These concern (1) involvement of mGluRl, (2) involvement of glutamate receptor δ2 subunit (GluRδ2), (3) involvement of Ca2+ release from intracellular stores, and (4) origin of nitric oxide (NO). I would like to add these new results to the review article by Crépel et al. [CRÉPEL et al.]