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What becomes of young people who display strong psychopathy traits? By combining cutting-edge research with interviews from over 500 incarcerated youth assessed for psychopathy and involved in serious, violent offenses, this book investigates whether they are destined to persist in crime throughout their lives. Evan McCuish explores not only long-term offending patterns but also psychopathy's influence on relationships, employment, substance use, and mortality. Through this, the text clarifies the meaning of the clinical construct of psychopathy and debunks myths and misconceptions popularised by the true crime genre. This allows readers to more reliably interpret the accuracy of popular culture descriptions of psychopathy. Synthesising over 100 years of research, this book defines psychopathy and contributes new knowledge to the field. It is ideal for students, scholars, and practitioners in psychology, criminology, social work, and law seeking further insight into this intriguing disorder.
The individual effects of genetic factors and adverse childhood experiences (ACEs) on risk of psychosis, including schizophrenia (SCZ) and bipolar disorder (BIP), have been widely acknowledged, but their interaction effects on individual psychopathological symptoms remain unclear.
Methods
Based on data from 163,704 individuals in the UK Biobank, we investigated the joint effects of polygenic risk scores (PRSs) of SCZ and BIP and ACEs on psychopathology. ACEs status and 55 psychopathological symptoms from seven domains were measured retrospectively using an online mental health questionnaire in 2016. Recent genome-wide association studies for SCZ and BIP were combined with genotype data to generate PRSs. Logistic regression analyses were then conducted to explore univariate and joint main effects of PRSs and ACEs on psychopathological symptoms, as well as their additive and multiplicative interaction effects.
Results
The interaction mechanisms for PRSs and ACEs varied across symptom domains: additive interactions were observed on the depression (RERIBIP-ACEs = 0.20–0.25), anxiety (RERISCZ-ACEs = 0.20; RERIBIP-ACEs = 0.22–0.26), help-seeking (RERISCZ-ACEs = 0.24; RERIBIP-ACEs = 0.23), and cognition domains (RERISCZ-ACEs = −0.23 to -0.17), whereas multiplicative interactions were only detected on the psychotic (betaSCZ-ACEs = −0.543; betaBIP-ACEs = −0.181), mania (betaBIP-ACEs = −0.195), self-harm or suicide (betaSCZ-ACEs = −0.118), and cognitive domains (betaSCZ-ACEs = −0.204 to −0.157).
Conclusions
The interplay mechanisms for genetic liability to SCZ and BIP and ACEs vary across symptom domains. This study reveals heterogeneity in gene–ACEs interaction mechanisms underlying psychosis and may provide personalized guidance for psychological care after ACEs.
This chapter begins with the perspective that subject matter experts have failed to be public with their definitions of psychopathy, and as a consequence, the general public has a limited understanding of psychopathy as a clinical construct. This chapter describes the origins of the term psychopathy and the nonlinear path that has been taken to establish contemporary definitions of this construct. The chapter outlines four key perspectives of psychopathy: Cleckley’s perspective, the triarchic model of psychopathy, the comprehensive assessment of psychopathy personality, and the view of psychopathy traits as extreme manifestations of general personality traits. The goal is not to convince readers which perspective they should adopt, but rather to help clarify similarities and differences so that the meaning of psychopathy can be more readily understood depending on the context in which it is being used. Indeed, different contemporary perspectives on psychopathy tend to share in common the view that core psychopathy traits emerge from interpersonal, affective, and behavioural domains of functioning.
This chapter brings to light the salience of psychopathy and personality disorders to cult leaders and their followers. Cult leaders present with distinctive personalities that enable them to exert inordinate influence over others and to exhibit highly manipulative and remarkably destructive conduct. Within the literature on cults, narcissistic and antisocial personality disorders are among the most commonly identified character pathologies ascribed to cult leaders. However, psychopathy may be the most fitting yet least explored constellation of traits and behaviors observed in such individuals. This chapter sheds light on the neglected perspective of psychopathic cult leaders while also focusing on the Cluster B personality disorders. The corresponding psychological profile that emerges for cult followers is typified by a noteworthy comorbidity of antisocial, obsessive compulsive, and dependent personality disorders. Suggestive forensic instruments to utilize for the clinical and forensic assessment of cult leaders and their followers include the PCL-R, Rorschach, MMPI-2, and TRAP-18.
This chapter of the handbook inspects the relationship between antisociality and morality from the dual perspectives of moral psychology and moral neuroscience. The authors provide a comprehensive overview of research on the moral cognition of different types of antisocial individuals. Based on their review of the research, they suggest that the capacity for moral reasoning in psychopathy is less defective than generally assumed. While the propensity of psychopathic individuals to engage in immoral behavior is due largely to affective deficits, it also stems from dysfunction in the neural circuitry underlying moral decision making. This simple narrative, however, is complicated by the fact that there is no single explanation of the immoral behavior exhibited by the full range of antisocial individuals. For example, while dysfunction in the neural circuitry of moral decision making may account for the immoral behavior of individuals with primary psychopathy and individuals prone to proactive aggression, it is less apt for explaining similar behavior by individuals with secondary psychopathy and a propensity for reactive aggression.
Antisocial personality disorder (ASPD) is a mental condition in which a person exhibits a pattern of repeated disregard for and violation of others’ rights. The Diagnostic and Statistical Manual of Mental Disorders (DSM) provides the framework for diagnosing ASPD. The prevalence rate of ASPD is high in prisons. Genetics, epigenetics, neuroscience, sociology, epidemiology, psychology, and other behavioral science fields have attempted to find a primary etiology. Despite decades of research, the precise etiology of ASPD has not been found, and its pathophysiology remains a complex question. The interaction between genes and the environment appears to be a significant factor in the development of ASPD. Brain imaging in subjects with ASPD has revealed structural brain changes in those with ASPD. Individuals with ASPD are challenging to manage for health care providers because of their complicated clinical presentation, high comorbidity of medical and mental disorders, lack of licensed pharmacotherapies for ASPD, and increased utilization of healthcare resources. ASPD requires tremendous effort from treating clinicians. Clinicians can successfully manage individuals with ASPD if they remain aware of the unique challenges of these patients while thoughtfully applying available research.
Altered affective state recognition is assumed to be a root cause of aggressive behavior, a hallmark of psychopathologies such as psychopathy and antisocial personality disorder. However, the two most influential models make markedly different predictions regarding the underlying mechanism. According to the integrated emotion system theory (IES), aggression reflects impaired processing of social distress cues such as fearful faces. In contrast, the hostile attribution bias (HAB) model explains aggression with a bias to interpret ambiguous expressions as angry.
Methods
In a set of four experiments, we measured processing of fearful and angry facial expressions (compared to neutral and other expressions) in a sample of 65 male imprisoned violent offenders rated using the Hare Psychopathy Checklist-Revised (PCL-R, Hare, R. D. (1991). The psychopathy checklist–revised. Toronto, ON: Multi-Health Systems) and in 60 age-matched control participants.
Results
There was no evidence for a fear deficit in violent offenders or for an association of psychopathy or aggression with impaired processing of fearful faces. Similarly, there was no evidence for a perceptual bias for angry faces linked to psychopathy or aggression. However, using highly ambiguous stimuli and requiring explicit labeling of emotions, violent offenders showed a categorization bias for anger and this anger bias correlated with self-reported trait aggression (but not with psychopathy).
Conclusions
These results add to a growing literature casting doubt on the notion that fear processing is impaired in aggressive individuals and in psychopathy and provide support for the idea that aggression is related to a hostile attribution bias that emerges from later cognitive, post-perceptual processing stages.
This Element offers a framework for exploring the methodological challenges of neuroethics. The aim is to provide a roadmap for the methodological assumptions, and related pitfalls, involved in the interdisciplinary investigation of the ethical and legal implications of neuroscientific research and technology. It illustrates these points via the debate about the ethical and legal responsibility of psychopaths. Argument and the conceptual analysis of normative concepts such as 'personhood' or 'human agency' is central to neuroethics. This Element discusses different approaches to establishing norms and principles that regulate the practices addressed by neuroethics and that involve the use of such concepts. How to characterize the psychological features central to neuroethics, such as autonomy, consent, moral understanding, moral motivation, and control is a methodological challenge. In addition, there are epistemic challenges when determining the validity of neuroscientific evidence.
Successful leaders are at risk of developing exaggerated pride, contempt for others, and a diminished sense of reality. The ancient Greeks feared this syndrome and called it hubris. Although certain contemporaneous leaders show signs of hubris and pose a great danger, the hubris syndrome does not yet figure in our classification systems. The purpose of this paper is to examine several aspects of its validity, including clinical description, laboratory study, and exclusion of other disorders. Firstly, a substantial body of evidence indicates that the hubris syndrome may develop after a person has held substantial power for a considerable amount of time. Thus, the syndrome differs from a personality disorder with its characteristic onset in late adolescence or early adulthood. It is proposed, therefore, that the syndrome is a non-organic personality change after gaining substantial power or achieving overwhelming success, characterized by the emergence or marked increase of pathological personality traits within the domains of dissociality and disinhibition. Within the domain of dissociality, grandiosity is an obligatory trait. Secondly, with reference to laboratory study, recent evidence suggests that machine learning algorithms have the ability to differentiate hubristic from non-hubristic speech patterns. Thirdly, the exclusion of other disorders is difficult, because individuals with the hubris syndrome do not collaborate in any investigation. Some suggestions are made to overcome this problem. In conclusion, there is sufficient reason to further examine the validity of the hubris syndrome and to consider it for inclusion in our classification systems.
This chapter describes pseudoscience and questionable ideas related to antisocial personality disorder. The chapter opens by considering diagnostic controversies such as the construct of psychopathy. Questionable assessment practices and myths that influence treatment are also addressed. Dubious treatments include psychedelics, psychoanalysis, and punishment-based interventions. The chapter closes by reviewing research-supported approaches.
The cluster of psychiatric concepts that includes “personality disorders,” “psychopathy” and “moral insanity” has long been controversial and uncertain. This article investigates the concept of “psychopathy” in 1950s England and shows how this ambiguity is not a flaw or failure in the concept but absolutely necessary for the role it carries out: policing broad areas of social life. A case of Munchausen syndrome (a type of “psychopathy”) in the late 1950s still functions as a precedent in the welfare system today, denying claimants sickness benefit, “closing a loophole,” and exemplifying the usefulness of this uncertainty.
Although the primary aim of Just as Deadly is to present information on female serial killers (FSKs), the author discusses a comparison of FSKs and male serial killers (MSKs) to illustrate the dramatic differences between their crimes. As described in Evolutionary Behavioral Sciences, the author and her team investigated sex differences in serial murder through an evolutionary psychological lens but also collected data on other background and mental health variables, as empirical comparisons of FSKs and MSKs are virtually nonexistent in the scientific literature. Data collected from 55 MSKs and 55 FSKs revealed sex differences in relationship status, educational attainment, and socioeconomic status. Moreover, nearly 90% of MSKs and 43% of FSKs had reported mental health issues. These results show that MSKs have more than a fourfold chance of having a mental illness compared to the population and are twice as likely as FSKs to have documented issues. The cases of MSKs Ed Gein and John Wayne Gacy describe mental illness issues and are used to demonstrate drastic differences in serial murder modus operandi and victims.
A single perspective cannot explain the entirety of serial murder. In psychology, we promote a biopsychosocial model of understanding any behavior or mental process. The author provides a summary of various perspectives, starting with behavioral neuroscience (i.e., biopsychology). She summarizes direct evidence from case studies showing neural issues in serial murderers, implied evidence of brain dysfunction of serial murders, and evidence of no neural issues in serial murderers. She then discusses the biological correlates of violence in general. The case of MSK Joseph DeAngelo highlights crime solved through forensic genetic genealogy.
Several scholars have recognized the limitations of theories of moral reasoning in explaining moral behavior. They have argued that moral behavior may also be influenced by moral identity, or how central morality is to one’s sense of self. This idea has been supported by findings that people who exemplify moral behavior tend to place more importance on moral traits when defining their self-concepts (Colby & Damon, 1995). This paper takes the next step of examining individual variation in a construct highly associated with immoral behavior — psychopathy. In Study 1, we test the hypothesis that individuals with a greater degree of psychopathic traits have a weaker moral identity. Within a large online sample, we found that individuals who scored higher on a measure of psychopathic traits were less likely to base their self-concepts on moral traits. In Study 2, we test whether this reduced sense of moral identity can be attributed to differences in moral judgment, which is another factor that could influence immoral behavior. Our results indicated that the reduced sense of moral identity among more psychopathic individuals was independent of variation in moral judgment. These results suggest that individuals with psychopathic traits may display immoral behavior partially because they do not construe their personal identities in moral terms.
The psychological correlates of utilitarian choices in sacrificial moral dilemmas are contentious. In the literature, some research (Greene, et al., 2001) suggested that utilitarianism requires analytic thinking while other research (Kahane et al., 2015) showed that utilitarianism is correlated with psychopathy. In the present research, we looked at the relation of several normative views with analytic cognitive style (ACS), psychopathy and real-world utilitarianism in three Turkish samples. In Study 1 (n = 269), we used four ethical dilemmas and asked participants to select one normative principle as the grounds for their judgment in the dilemma: fatalism, virtue ethics, utilitarianism, deontology and amoralism. The results showed that the majority selected the deontological principle. Additionally, there was a considerable amount of fatalistic and virtue ethical justifications. Utilitarianism and psychopathy had a significant positive correlation. In Study 2 (n = 246), we replicated Study 1 and showed a significant relation between ACS and moral minimalism (the view that the sacrificial act is permissible but not necessary). In Study 3, the results showed that the utilitarian option in the sacrificial dilemmas was positively correlated with both real-life utilitarianism and psychopathy, but the latter two variables were not correlated with each other. All in all, the results suggest that some people choose the utilitarian option in moral dilemmas from psychopathic tendencies (as Kahane argued), while others due to real-life utilitarian reasons (as Greene argued). The findings also indicate that virtue ethical and fatalistic justifications cannot be ignored in understanding lay people’s moral judgments.
In response to arguments made by Gawronski et al. (2020), responding to Baron and Goodwin (2020), we concentrate on four issues. First, the CNI design requires substantial numbers of “perverse” responses to congruent items — those in which both consequences and norms both favor action, or both favor inaction — and these responses depend on the ambiguity of the items concerning which norms apply or which consequences are worse. Effects of outside variables, such as psychopathy, may result from the effect of such variables on the interpretation of ambiguous items, rather than from their effect on sensitivity to norms or consequences. Second, the CNI design may not be so useful at measuring general action/inaction biases. Third, the order of the two processes in the model could in fact affect the conclusions drawn (even though it does not do so in most studies done so far). Fourth, the conclusions drawn do in fact depend on the items’ susceptibility to reinterpretation (owing to their ambiguity): the tests proposed for item validity are too weak, since they require only that a majority of subjects agree with the experimenters’ classification, even though a minority could affect the conclusions drawn. We illustrate some of our points with an analysis of the psychopathy study of Luke and Gawronski (2020).
The study of psychopathic traits has increased in recent years, given the impact that these traits have on our society.
Objectives
This study aimed to evaluate the relationship between psychopathy traits and ability emotional intelligence by examining the sub-dimensions of both constructs in a sample of incarcerated males.
Methods
A total of sixty-three incarcerated adult males (Mage = 37.51) were assessed for psychopathy traits and emotional intelligence levels through the 34-item Self-Report Psychopathy Scale-III (SRP-III) and the Mayer-Salovey-Caruso Emotional Intelligence Test (MSCEIT) respectively.
Results
The results revealed that the incarcerated population is characterized by low EI and high psychopathic traits (explained by the scores obtained on the criminal tendencies sub-dimension). Moreover, participants scoring lower in ability EI were more likely to score higher on the callous affect sub-dimension of psychopathy. We also observed an indirect negative effect of ability EI on erratic lifestyle, criminal tendencies and interpersonal manipulation sub-dimensions through the mediating role of callous affect.
Conclusions
These findings offer a better understanding of the relationship between psychopathy traits and ability emotional intelligence and provide empirical support for the need to implement intervention programs in penitentiary centers based on EI training, which could help to reduce antisocial and disruptive behaviours and facilitate future reintegration into society.
Although psychopathy is widely conceptualised as a mental disorder, some researchers question the maladaptive nature of psychopathy, and argue that it might be advantageous from an evolutionary point of view. According to this view, psychopathy can be seen as an evolutionary adaptative strategy that relies on deception and manipulation to gain short-term reproductive benefits. Psychopathy is also identified as a fast life strategy in response to early life stress and an adaptation to harsh environments. This paper investigates the evidence that psychopathic traits are adaptive, while also addressing the limitations of current evolutionary models of psychopathy based on frequency-dependent selection and life history theory. We review recent studies on the fitness correlates of psychopathy and find that psychopathic traits present potential adaptive trade-offs between fertility and mortality, and offspring quantity and quality. On a proximate level, individual differences in stress reactivity and environmental risk factors in early development predispose to psychopathy through gene–environment interactions. We propose that environmental, developmental, social and cultural factors can mediate the relationship between psychopathic traits and fitness and therefore should be considered to make accurate predictions on the adaptive potential of psychopathy. We end by outlining gaps in the literature and making recommendations for future evolutionary research on psychopathy.
People are susceptible to boredom and seek an optimal level of arousal. Psychopathic individuals appear to be low on arousal, and they try to elevate this by seeking novelty and taking risks. Ways to achieve the optimal level by sensation-seeking include transgressions such as violence, arson and theft. These can have sexual associations and are sometimes accompanied by masturbation. Dopamine activation is at the basis of elevations in arousal. Serial lust killers commonly have a history of committing non-sexual crimes and arson. The internal organs of the body are under the control of the autonomic nervous system, high arousal being associated with dominance of the sympathetic branch and low arousal with the parasympathetic branch. Arousal is largely non-specific, and it takes a positive or negative value according to context. Switches can be made from negative to positive, which appears to lie at the basis of some lust killing.