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Psychologists and psychological research have shaped sleep and circadian science for over a century. Yet, psychology has not fully embraced sleep as a core area of inquiry, and sleep medicine has not distinctly acknowledged the foundational role psychology plays in understanding sleep and circadian rhythms. This Question Paper invites submissions exploring psychology’s profound impact on the study, measurement and intervention strategies in sleep and circadian science, as well as reciprocal influences. Manuscripts may include historiographies of key contributors, laboratory milestones, theoretical advancements and methodological innovations within a historical context. We aim to capture the full scope of sleep psychology from its origins to a vision of its future.
Late Life Major Depressive Disorder (LLD) and Hoarding Disorder (HD) are common in older adults with prevalence estimates up to 29% and 7%, respectively. Both LLD and HD are characterized by executive dysfunction and disability. There is evidence of overlapping neurobiological dysfunction in LLD and HD suggesting potential for compounded executive dysfunction and disability in the context of comorbid HD and LLD. Yet, prevalence of HD in primary presenting LLD has not been examined and potential compounded impact on executive functioning, disability, and treatment response remains unknown. Thus, the present study aimed to determine the prevalence of co-occurring HD in primary presenting LLD and examine hoarding symptom severity as a contributor to executive dysfunction, disability, and response to treatment for LLD.
Participants and Methods:
Eighty-three adults ages 65-90 participating in a psychotherapy study for LLD completed measures of hoarding symptom severity (Savings Inventory-Revised: SI-R), executive functioning (WAIS-IV Digit Span, Letter-Number Sequencing, Coding; Stroop Interference; Trail Making Test-Part B; Letter Fluency), functional ability (World Health Organization Disability Assessment Schedule-II-Short), and depression severity (Hamilton Depression Rating Scale) at post-treatment. Pearson's Chi-squared tests evaluated group differences in cognitive and functional impairment rates and depression treatment response between participants with (HD+LLD) and without (LLD-only) clinically significant hoarding symptoms. Linear regressions were used to examine the association between hoarding symptom severity and executive function performance and functional ability and included as covariates participant age, years of education, gender, and concurrent depression severity.
Results:
At post-treatment, 24.1% (20/83) of participants with LLD met criteria for clinically significant hoarding symptoms (SI-R.41). Relative to LLD-only, the LLD+HD group demonstrated greater impairment rates in Letter-Number Sequencing (χ2(1)=4.0, p=.045) and Stroop Interference (χ2(1)=4.8, p=.028). Greater hoarding symptom severity was associated with poorer executive functioning performance on Digit Span (t(71)=-2.4, β=-0.07, p=.019), Letter-Number Sequencing (t(70)=-2.1, β=-0.05, p=.044), and Letter Fluency (t(71)=-2.8, β=-0.24, p=.006). Rates of functional impairment were significantly higher in the LLD+HD (88.0%) group compared to the LLD-only (62.3%) group, (χ2(1)=5.41, p=.020). Additionally, higher hoarding symptom severity was related to greater disability (t(72)=2.97, β=0.13, p=.004). Furthermore, depression treatment response rates were significantly lower in the LLD+HD group at 24.0% (6/25) compared to 48.3% (28/58) in the LLD-only group, χ2(1)=4.26, p=.039.
Conclusions:
The present study is among the first to report prevalence of clinically significant hoarding symptoms in primary presenting LLD. The findings of 24.1% co-occurrence of HD in primary presenting LLD and increased burden on executive functioning, disability, and depression treatment outcomes have important implications for intervention and prevention efforts. Hoarding symptoms are likely under-evaluated, and thus may be overlooked, in clinical settings where LLD is identified as the primary diagnosis. Taken together with results indicating poorer depression treatment response in LLD+HD, these findings underscore the need for increased screening of hoarding behaviors in LLD and tailored interventions for this LLD+HD group. Future work examining the course of hoarding symptomatology in LLD (e.g., onset age of hoarding behaviors) may provide insights into the mechanisms associated with greater executive dysfunction and disability.
The Arabian leopard Panthera pardus nimr is categorized as Critically Endangered, with < 200 individuals estimated to remain in the wild. Historically the species ranged over an extensive area of western Saudi Arabia but, with no confirmed sightings since 2014, investigating potential continued presence and distribution is of critical conservation importance. We present the results of a comprehensive survey designed to detect any remaining Arabian leopard populations in Saudi Arabia. We conducted 14 surveys, deploying 586 camera-trap stations at 13 sites, totalling 82,075 trap-nights. Questionnaire surveys were conducted with 843 members of local communities across the Arabian leopard's historical range to assess the presence of leopards, other predators and prey species. Predator scats were collected ad hoc by field teams and we used mitochondrial DNA analysis to identify the originating species. We obtained 62,948 independent photographs of animals and people, but none were of Arabian leopards. Other carnivores appeared widespread and domestic animals were numerous, but wild prey were comparatively scarce. Three questionnaire respondents reported sightings of leopards within the previous year, but targeted camera-trap surveys in these areas did not yield evidence of leopards. Of the 143 scats sent for analysis, no DNA was conclusively identified as that of the leopard. From this extensive study, we conclude there are probably no surviving, sustainable populations of Arabian leopards in Saudi Arabia. Individual leopards might be present but were not confirmed. Any future Arabian leopard conservation in Saudi Arabia will probably require reintroduction of captive-bred leopards.
Improving the quality and conduct of multi-center clinical trials is essential to the generation of generalizable knowledge about the safety and efficacy of healthcare treatments. Despite significant effort and expense, many clinical trials are unsuccessful. The National Center for Advancing Translational Science launched the Trial Innovation Network to address critical roadblocks in multi-center trials by leveraging existing infrastructure and developing operational innovations. We provide an overview of the roadblocks that led to opportunities for operational innovation, our work to develop, define, and map innovations across the network, and how we implemented and disseminated mature innovations.
Recent arguments claim that behavioral science has focused – to its detriment – on the individual over the system when construing behavioral interventions. In this commentary, we argue that tackling economic inequality using both framings in tandem is invaluable. By studying individuals who have overcome inequality, “positive deviants,” and the system limitations they navigate, we offer potentially greater policy solutions.
We expand Chater & Loewenstein's discussion of barriers to s-frames by highlighting moral psychological mechanisms. Systemic aspects of moralized social issues can be neglected because of (a) the individualistic frame through which we perceive moral transgressions; (b) the desire to punish elicited by moral emotions; and (c) the motivation to attribute agency and moral responsibility to transgressors.
One challenge for multisite clinical trials is ensuring that the conditions of an informative trial are incorporated into all aspects of trial planning and execution. The multicenter model can provide the potential for a more informative environment, but it can also place a trial at risk of becoming uninformative due to lack of rigor, quality control, or effective recruitment, resulting in premature discontinuation and/or non-publication. Key factors that support informativeness are having the right team and resources during study planning and implementation and adequate funding to support performance activities. This communication draws on the experience of the National Center for Advancing Translational Science (NCATS) Trial Innovation Network (TIN) to develop approaches for enhancing the informativeness of clinical trials. We distilled this information into three principles: (1) assemble a diverse team, (2) leverage existing processes and systems, and (3) carefully consider budgets and contracts. The TIN, comprised of NCATS, three Trial Innovation Centers, a Recruitment Innovation Center, and 60+ CTSA Program hubs, provides resources to investigators who are proposing multicenter collaborations. In addition to sharing principles that support the informativeness of clinical trials, we highlight TIN-developed resources relevant for multicenter trial initiation and conduct.
Breathing is a function so fundamental to life that most of the time its significance goes unrecognised; yet the absence of the ability to breathe freely is a reality for many. The recent global COVID-19 pandemic – which particularly affects the lungs – has exposed an intersection of pandemic and capitalism in which some bodies are affected more than others; research findings show that people of the global majority have been disproportionately impacted by coronavirus (University of Manchester, 2020). Simultaneously, the death of George Floyd, and the pain of his final words, ‘I can't breathe’, has shown how the life-breath of some humans is more valued than that of others.
This chapter draws on bell hooks’ (1994: 130) notion of critical dialogue; a practice that involves ‘individuals who occupy different locations … mapping out terrains of commonality, connection and shared concern’. For hooks coming together in the spirit of beloved community requires us to cross borders, thereby enriching each other's knowledge. As educators at either ends of the spectrum of privilege and oppression (Author A is a white female academic, and Author B an activist-writer of colour), we have used this dialogic space to explore the intersection of pandemic and inequality. By making the choice to write anonymously, we have also made a deliberate effort to detach from the ego associated with academic writing, and we invite others to adopt a similar dialogic model by which to converse as equals (privilege and structural inequalities notwithstanding).
We begin from a series of questions: Who is able to breathe? Why are some individuals afforded ‘breathing space’ and not others? What can we learn from the necropolitics of the pandemic (Mbembe and Corocoran, 2019), and how might we utilise this knowledge to take action for affirmative social change in education? We then explore the role of education in making change and conclude by reflecting on the role of dialogic processes in furthering and enriching understanding.
Whether denied, derided or determined to overcome it, COVID-19 has impacted many lives in ways that we are only now beginning to witness, as we move from old configurations of normality and adapt to new realities, be it flexible ways of working and learning or working to change social systems. It is also evident that COVID-19 extends beyond a global health problem. Sociocultural readings of the pandemic have pointed to this as a crisis on multiple levels – economic, environmental, social, cultural and racial. It, then, becomes an important task to understand how this crisis has affected, and continues to affect, people across the world, through readings that do not lean into othering and moralism, as is often the main societal response, and as we have seen throughout this text.
With no end yet in sight, COVID-19 and its variants – from the South African variant to Omicron then to Omicron BA.4 to BA.5 – are still widespread and affecting the lives and health of many people. And amid the political and social turmoil of summer 2022, as the populace contends with strikes by railway workers and barristers, the cost of living crisis and the political turbulences of the British government, we must remember and be ever vigilant of the impact of the rampant racism evident in our society. In the early days of the pandemic, the COVID variants were not referred to by scientific nomenclature but by the site of origin – for example COVID-19's origins in China, then the South African strain, then the Indian strain. This associated racialisation of a virus was intriguing to observe, since it resurrected and reinforced the trope of dirty unclean foreigner/outsider/subaltern, and drew implicit links between biology and race. The naming of the variants by country of origin was eventually dropped in favour of scientific nomenclature. This may have occurred as someone, somewhere, realised that the links between the easy and lazy association of the new variant by country of origin could reinforce certain racial stereotypes. But the damage had already been done. So, while at the same time living with and suffering from the virus, Black and global majority people have suffered insulting microaggressions associated with the early identification of the COVID-19 virus variants, and simultaneously been lost to the virus in greater numbers. The saying ‘adding insult to injury’ jumps to mind.
On 31 December 2019, the world began to hear murmurs from the Chinese authorities and the World Health Organization (WHO) of the novel coronavirus, a mysterious new virus strain that had never been identified in humans. By 30 January 2020, WHO labelled SARS-Cov-2 (COVID-19) as a public health emergency of international concern. It did not take long (11 March 2020) for the widespread and rapidly evolving virus to be declared, a global pandemic.
If a pandemic is defined as ‘a novel infection – new and previously unconfronted – that spreads globally and results in a high incidence of morbidity (sickness) and mortality (death)’, deriving ‘from pan – across, and demos, meaning people or population’ (Doherty, 2013: 42), the new virus in this instance became confronted with an old one that has historical roots and continual contemporary manifestations – and that is the virus of racism. The antagonism, discrimination and prejudice directed against an individual of another race based on one's perceived racial superiority is not novel by any means. However, the histories of racism in the global North and South have never been more visible than in these pandemic times. Indeed, the impact of the latter on existing inequalities is wide-reaching, with reverberations felt worldwide. Even after three years since the arrival of COVID-19, many of the outcomes from these inequalities have been shown to be more exacerbated and devastating, particularly along the lines of race and ethnicity.
Global media discourses and scholarly debates have revealed over the course of 2020 onwards the role structural racism has played in perpetuating racial inequalities, and its impacts on Black and global majority groups has been laid bare. The COVID-19 pandemic has been a catalyst of many existing and intersectional issues globally, and it was with this in mind that the Centre for Race, Education and Decoloniality (CRED) at Leeds Beckett University initially issued a call for rapid response papers on the racial impacts of the coronavirus. More widely, we identified an emerging racist rhetoric of blaming and shaming Black, Asian and Minority ethnic groups, for instance in the conversations on vaccine hesitancy, higher COVID-19 incidence in Black communities, the labelling of the virus as the ‘Chinese virus’ and the ‘Indian variant’, to enumerate just a few examples.
For decades, symbolic models of cognition were the dominant computational approaches of cognition. Today they coexist with subsymbolic, statistical, and hybrid models, but they are still the de facto standard for modeling human reasoning processes. This chapter summarizes important aspects of symbolic and hybrid models of cognition, approaching the topic from different perspectives. After some discussion on historical aspects and the theoretical basis of symbolic models of cognition, cognitive architectures as models for intelligent agents are examined. Subsequently, the role of symbolic computational approaches towards processing natural language, representation of human knowledge, and commonsense reasoning are considered. Then the focus is put on the crucial question of learning new representations and theories, before finally looking at hybrid and neural-symbolic systems combining reasoning and learning and bridging between symbolic and subsymbolic elements.