Parental mentalizing, or the parent’s capacity to think about the child as having an inner psychological world, has been shown to play an important role in sensitive parenting and child socioemotional development. Studies suggest that high levels of stress impair (parental) mentalizing, yet surprisingly few studies have experimentally investigated this. The present study aimed to address this gap by investigating the impact of child-focused stress on parental mentalizing measured using a newly developed self-report questionnaire, following an experimental design with a computer-controlled baby simulator in a sample of 29 community mothers. Both subjective arousal, measured by a self-report item, and biological arousal, assessed through galvanic skin response, were measured throughout the experiment. Attachment dimensions, childhood trauma, and borderline personality disorder (BPD) features were assessed at baseline. Results demonstrated that the induction of child-focused stress was associated with an increase in parental mentalizing difficulties. Increases in mentalizing difficulties were, in turn, associated with increases in subjective and biological arousal following the simulator task. Finally, attachment anxiety and childhood trauma were positively correlated with both arousal and parental mentalizing difficulties in the simulator task, whereas attachment avoidance and BPD features were not. The implications of these findings for early intervention are discussed.

Previous research has suggested bidirectional relations between depressive symptoms and both internal and external core beliefs (self-esteem and optimism, respectively) in adolescence. However, little work has examined the cultural commonality versus specificity of these developmental pathways in adolescence across diverse contexts. To address this gap, the current study traced bidirectional associations among depressive symptoms and two forms of core beliefs (self-esteem and optimism) in adolescents from 12 cultural groups in nine countries. Longitudinal data were collected from 1,090 adolescents at ages 15 and 17. Significant associations emerged between age 15 depressive symptoms and both age 17 core beliefs across all cultural groups except Sweden. No significant associations between age 15 core beliefs and age 17 depressive symptoms were found in the multigroup model. However, the pathways from core beliefs to depressive symptoms and from depressive symptoms to core beliefs did not significantly differ in strength. These findings provide cross-cultural evidence for the scar theory (depressive symptoms → core beliefs), but no clear support for the vulnerability theory (core beliefs → depressive symptoms), perhaps due to the measurement and stability of depression. These findings have implications for understanding the adolescent development of psychopathology and cognitions, such as core beliefs, across diverse cultures.

Despite the growing body of research on the intergenerational transmission of problem behavior, there is a need for more integrative approaches that consider the interplay between genetic and environmental factors. This study uses unique longitudinal data from TRAILS (analytic sample n = 2202), a prospective multiple-generation cohort study in the Netherlands to examine whether parents’ problem behavior (parents’ self-reported lifetime antisocial behavior and substance use, reported at mean age 40 years) predicts offspring problem behavior nearly two decades later (offspring self-reported aggression and delinquency at mean ages 29 and 32 years). In path analyses, independent and relative contributions of genetic (polygenic scores of parents and offspring) and environmental (harsh parenting) pathways were tested. Results confirm intergenerational transmission and consistently point to genetic nurture whereby genetic predisposition predicts parental problem behavior, which in turn predicts harsh parenting, which in turn predicts offspring problem behavior, all while accounting for offspring genetic predisposition, sex and family socioeconomic position. Though these findings are surprising in light of genetic contributions to behavior, they allow for tentative considerations regarding implication for practice to help reduce the continuation of problem behaviors across generations.

The quality of research across psychology needs improvement. Ample evidence has indicated that publication bias, specifically making publication decisions based on a study’s results, has led to a distorted literature (e.g., high rates of false positives). Registered Reports, which can now be submitted to Development and Psychopathology, are a recent publication format designed to combat publication bias and problematic research practices. The format represents a shift from a system in which publication decisions are based on the nature of the findings, to one that is based on the quality of the study conceptualization and design. In this invited Views article, we introduce the Registered Reports format to Development and Psychopathology by arguing that they can and should be used in developmental psychopathology research. We first describe what Registered Reports are and why they are useful. We then review 10 commonly expressed concerns about publishing Registered Reports – including that they are not appropriate for studies using preexisting data, that they do not allow for exploratory analyses, and that they take too long to publish – explaining why these concerns are unwarranted. We hope that this article will allay concerns about publishing Registered Reports, and that readers will submit them to Development and Psychopathology.

Refugee youth are at high risk for trauma-related disorders – outcomes not only the result of pre-migration trauma, but consequences of diverse post-migration stressors. This study identified individual, parental, and environmental factors – some potentially modifiable – associated with trajectories of psychological risk and resilience in 291 Syrian and Iraqi refugee youth during resettlement in the U.S. Data was collected at arrival and at two follow-up visits up to 7 years post-arrival. Linear mixed modeling assessed predictors of posttraumatic stress disorder (PTSD), anxiety, and depression trajectories. Victimization trauma (i.e., assault) and lower maternal subjective social status predicted more severe PTSD (p = .046, f 2 = .07; p < .001, f 2 = .23) and anxiety (p = .008, f2 = .05; p = .002, f 2 = .11) trajectories in youth. Paternal unemployment predicted less stable PTSD (p = .009, f2 = .13) and anxiety (p < .001, f 2 = .10) trajectories. More severe depression trajectories were associated with female sex (p = .045, f 2 = .06) and death threat traumas (p = .014, f 2 = .07). Findings identified predictors of long-term risk and resilience for refugee youth, as well as potentially modifiable ecological risk factors. Victimization and death threat trauma exposure could be salient in identifying youth at high risk for trauma-related symptoms early in resettlement. Indicators of financial security were also associated with symptoms, suggesting environmental intervention targets.

Although depression can be transmitted across generations, less is known about how this cycle can be interrupted. This study examines whether the multilevel Fast Track intervention (clinicaltrials.gov, NCT01653535) disrupts intergenerational transmission of depression. Children at high risk for aggression were randomly assigned to a 10-year control group or intervention targeting parenting and children’s intrapersonal, interpersonal, and academic skills. The original sample included 891 first-generation (G1) participants who reported on their depression and their children’s (second-generation; G2) internalizing problems. At age 34, 374 G2 participants (n = 191 intervention, n = 183 control) reported on their and their children’s (third-generation; G3) emotional difficulties. Mediated path models showed that a cascading model where higher G1 depressive symptoms influence higher G2 childhood depressive symptoms, leading to higher G2 adulthood depressive symptoms, which in turn is connected with greater G3 emotional difficulties, emerged only in the control group. The Fast Track intervention disrupted the pathways from G1 depressive symptoms to G3 emotional difficulties, from G2 childhood depressive symptoms to G2 adulthood depressive symptoms, and from G2 adulthood depressive symptoms to G3 emotional difficulties, highlighting the importance of preventive interventions in altering developmental trajectories of psychopathology.

Trust judgments involve rapidly evaluating others’ appearance and are critical in psychosocial development. Trust biases may be linked to psychopathology risk, particularly in vulnerable, adversity-affected populations, but very little is known about trust perception in refugee context. Here, we measured trust perception of Syrian refugee children (N = 324, Mage = 6.32 years) displaced in Jordan, using a validated trust task with computer-generated faces varying in perceived trustworthiness (data collection: May-August 2021). Mothers (N = 324, Mage = 32.59) reported on child and mothers’ mental health, and mother-child relationship. Child trust perception was not associated with child or mothers’ mental health, or mother-child relationship (all p > .10), but we found age-related changes in perceived trust, with older children reporting faces as less trustworthy than younger children (B = .32, p < .001). Although children’s social judgments might be associated with socio-emotional functioning in non-refugee populations, our results suggest that refugee children’s mental health does not seem to be linked to their perception of trust, and that trusting others might diminish with age in displaced, at-risk children.

The current study examined how early smartphone ownership impacts parent-child informant discrepancy of youth internalizing problems during the transition to adolescence. We used four waves of longitudinal data (Years 1–4) from the Adolescent Brain Cognitive Development (ABCD; Baseline N = 11,878; White = 52.0%, Hispanic = 20.3%, Black = 15.0%, Asian = 2.1%, Other = 10.5%; Female = 47.8%). Across the full sample, significant parent-child informant discrepancy, such that parents underestimated child reports, appeared at Year 2 (Mage = 12.0) and increased across the remainder of the study (b = −0.21, SE = .042, p < .001, 95%CI [−.29, −.23]). Further, multi-group models indicated that significant parent-child informant discrepancy emerged in the years following initial smartphone acquisition, whereas youth who remained non smartphone owners did not demonstrate such a pattern. Moreover, this discrepancy grew with additional years of smartphone ownership. This study contributes to the ongoing discourse on adolescent smartphone use and mental health by documenting a novel, longitudinally observed risk to timely parental detection of mental health problems by early smartphone ownership.

Individuals with childhood experience of out-of-home care (OHC) face elevated risks of criminal behavior and poor mental health compared with the majority population. Evidence on how trajectories of offending and psychiatric disorders covary among individuals with experience of OHC is needed. This study is based on a cohort of 14,608 individuals (n = 1,319 with OHC experience) born in the Stockholm metropolitan area in 1953 (49% women) from birth to age 63 (2016). Group-based multi-trajectory modeling among those with at least one offense or psychiatric disorder (40.5% of the men, 16.6% of the women) identified four co-occurring trajectories for both sexes. Multinomial regression analyses showed that adolescent OHC placement, particularly in institutions and for behavioral reasons, was linked to higher odds of early-adulthood-limited or decreasing offending and psychiatric trajectories. Most individuals recover from offending and psychiatric disorders by retirement, but placed individuals in particular remain at high risk for offending, alongside psychiatric disorders, throughout early adulthood. Early assessment and tailored attention to needs and risk levels is important when designing long-term care services to mitigate this. Research on underlying mechanisms, and on collaboration between the welfare, justice, and psychiatric care systems, can help to design effective intervention strategies and policies.

While empathy is often seen as a resilience factor, emotional resonance with others’ suffering may increase psychological vulnerability during mass trauma exposure, particularly in youth. Since the role of early empathy as a prospective risk factor remains understudied, we used a decade-long longitudinal design to examine whether empathic reactions in childhood predicted early adolescents’ internalizing (depression and anxiety) symptoms following the October 7th attack and the Israel–Hamas war. Empathic distress was assessed at age 1.5 years and age 3 years through observational tasks. Emotional empathy and internalizing symptoms were self-reported at age 11 years, before the war, and reported again after its outbreak. Findings showed substantial internalizing symptoms during the war, with 31% of participants exceeding the clinical cutoff for anxiety and 23% for depression. Non of the empathy measures predicted internalizing symptoms before the war. However, during the war, empathic distress at age 1.5 and emotional empathy at age 11 predicted internalizing symptoms, controlling for negative emotionality and prior internalizing symptoms. Path analysis also linked empathic distress at age 3 to internalizing symptoms during war. Findings suggest that early empathic reactions may increase vulnerability to internalizing symptoms during mass trauma but not in non-traumatic contexts, aligning with a diathesis-stress model. Understanding empathy’s role in risk and resilience can inform interventions for youth exposed to war.

It is well established that childhood adversity is associated with both negative physical and mental health outcomes. Recent research posits that 1) there may be developmental periods for which the effects of adversity are most influential on brain development and 2) abuse and neglect may be associated with different developmental mechanisms linking psychopathology. This study used seven years of longitudinal data to investigate how abuse and neglect during three developmental periods (early childhood, middle childhood, and adolescence), as well as chronicity of maltreatment across these developmental periods, are associated with young adult mental health outcomes (ages 20–21), and how changes in adolescent task-based functional connectivity during cognitive control (between ages 14–15 and 18–20 years) may mediate these associations. Hypothesized mediation models were tested via structural equation modeling (SEM). Significant indirect effects indicated that chronic abuse predicted higher depressive symptoms and higher substance use through stronger dACC–insula connectivity. In contrast, significant indirect effects revealed that neglect during adolescence predicted lower substance use and lower depressive symptoms through weaker dACC–insula connectivity. These results suggest that differential patterns of connectivity changes within the salience network during cognitive control may be associated with risk and resilience for future depression and substance use in young adulthood.

This study assesses the relation between screen time, problematic media use behaviors, and clinical concerns (internalizing and externalizing problems) and suicidal ideation and non-suicidal self-injury within race/ethnicity and sex in the Adolescent Brain Cognitive Development (ABCD) Study (youth aged 11 to 12; N = 10,052). Understanding behaviors around screens (problematic media use), rather than focusing on screen time alone is useful in guiding clinical recommendations. In this analysis, regression models indicated that problematic media use consistently predicted clinical concerns with a larger effect size than screen media use. When examining how problematic media use and screen media use related to clinical concerns along domains of race/ethnicity and sex, problematic media use was a more consistent predictor of clinical concerns than screen media use for almost every race/ethnicity (except American Indian/Alaska Native participants). Problematic media use was also a consistent predictor of clinical concerns for both males and females, with some difference in screen media use predictors. This study has implications for the utility of assessing screen media use in research on clinical concerns in youth, and further suggests that researchers and clinicians should consider behaviors around screens in addition to screen time itself when assessing for impact on mental health.

Suicide rates are increasing rapidly among Black children and adolescents, calling for novel approaches to understanding their unique risk factors. The Structural Racism and Suicide Prevention Systems Framework offers a new culturally responsive theory that structural racism is an underlying mechanism for disparities in suicide among ethnoracial marginalized youth. Thus, a deeper analysis of the intersection of racism and systems to better understand suicide risk and create more effective targeted interventions for Black youth is imperative. The current systematic review comprehensively evaluated and synthesized the empirical literature regarding the relationship between structural racism and suicide risk among Black youth. 17 studies from 3 database searches, published between 2013 and 2024 are presented. Results revealed a positive relationship between structural racism and suicidal thoughts and behaviors among Black youth. Systems that particularly facilitate the perpetration of racism toward Black youth include schools, criminal justice, and income inequality. Findings serve as a call to action to incorporate more socioecological models into suicide prevention research focused on Black youth. Understanding the depth and scope of how racism contributes to suicide risk provides key targets for prevention and intervention strategies that are specific to individuals belonging to this group at disparate risk for suicide.

Fathers have a unique and critical role in children’s development, but limited empirical studies have examined prenatal predictors of fathers’ parenting behaviors. Exposure to early life stressors may alter adult brain white matter fibers, especially in fibers supporting optimal cognitive and emotional functioning. As such, men with experiences of early life stressors, such as risky family environments, may enter parenthood with neurobiological differences that impact their ability to provide optimal parenting. Few studies focus on early life stressors on men’s prenatal neurobiology and subsequent parenting outcomes. This study of first-time fathers (n = 41; Mage = 31.81 years; 32% Hispanic; 32% White; 24% Asian American; 7% Black; 5% Multiracial) investigated whether risky family environments would be associated with prenatal white matter organization and postpartum parenting (infants’ Mage = 6.96 months). White matter organization was quantified through fractional anisotropy (FA), a measure of the directionality of the fibers within the tissue. Fathers reporting riskier family environments exhibited lower FA in white matter tracts like fornix and cingulum, which support connections between brain areas underlying memory and emotion regulation. Lower FA in these regions predicted less effective parenting postpartum. Findings provide insight into intergenerational transmission of family risk.

It is well-established that adolescents’ temperament trajectories predict future psychopathology. Less well understood is how temperament traits co-develop from adolescence to young adulthood. We characterized how youths’ trajectories of effortful control, frustration, affiliation, and shyness formed multi-trajectory groups and examined their associations with adulthood psychopathology and polygenic risk scores (PRS). Participants were drawn from a larger longitudinal cohort (N = 1412). Effortful control, frustration, affiliation, and shyness were measured four times from ages 10-23. Adulthood internalizing and externalizing problems were measured at ages 24–27. PRS for externalizing problems and major depressive disorder were calculated. Group-based multi-trajectory analyses showed that a five-group model fit best, including “high-risk” on all temperament traits, “undercontrolled” and exuberant, “low-risk” on all traits, “overcontrolled” and inhibited, and “low affiliation” groups that differed on both the levels and slopes of temperament traits over time. The undercontrolled group showed the highest, and overcontrolled the lowest, externalizing PRS scores. The high-risk group showed heightened scores on the depression PRS. We found specific linkages between the high-risk group and withdrawn/depressed symptoms and the high-risk and undercontrolled groups with externalizing problems. Findings shed light on developmental patterns of temperament in adolescence-to-adulthood and unique combinations of temperament trajectories with specific linkages to etiologic factors and psychopathology.

Empirical literature on the trajectory of task performance in children is currently scarce. Therefore, this study investigates both the developmental trajectory of flanker task performance in children and the association with the development of teacher-reported problem behavior. Five waves of flanker performance and behavioral and emotional problems were drawn from a large longitudinal sample of elementary school children in the Netherlands (1424 children, ages 7 to 12 years). Latent growth curve modeling (LGM) identified a piecewise decrease in flanker response time: the steepest decline was found from 7 to 9 years old. Boys had lower levels of response time at age 7 than girls. Children showed a linear decrease in behavioral and emotional problems over time. Parallel LGMs revealed that lower levels of initial flanker response time were associated with a stronger decrease in anxiety problems and oppositional defiant-related behavior. A faster decline in response time was associated with a faster decline in depression problems, attention deficit hyperactivity-, and oppositional defiant-related behavior. Results offer insight into the normative development of performance monitoring in childhood and the link between behavioral measures of performance monitoring and behavioral and emotional problems. Future research should focus on the directionality of the association between performance monitoring and psychopathology.

Research finds genetic predisposition for depression is associated with increases in depression across adolescence and adulthood. In turn, depressive symptoms in adolescence are associated with substance use. However, there has been modest examination of genetic predisposition for depression, growth in depressive symptoms, and substance use from late childhood through adolescence, and mostly in White samples. Also, psychosocial interventions can attenuate associations between genetic predisposition and psychopathology, a genotype by intervention (GxI) effect. We examined associations among polygenic risk for depression, growth in depressive symptoms from age 7 to 16, and substance use at age 16, as well as moderation by a family-based preventive intervention. Participants were African-ancestry (n = 154) and European-ancestry (n = 219) youth from the Early Steps Multisite Study, half of whom participated in the Family Check-Up intervention. A small polygenic by intervention effect was found on reductions in depressive symptoms for African-ancestry youth, and growth in depressive symptoms was positively associated with substance use at age 16. In sensitivity analyses, a small GxI effect was detected in European-ancestry youth on reductions in depressive symptom slopes from age 10 to 16. These findings highlight how early intervention can buffer genetic effects on depressive symptoms over time.

Supportive parenting experiences link to secure attachment development, and secure attachment in turn links to children’s emotional well-being. However, little is known whether child-factors, like their environmental sensitivity, moderate these associations for better or for worse. We used longitudinal data (three data waves spanning two years) from 614 children (Wave 1: Mage = 10.28; SDage = 0.58; 44% boys). At all waves, attachment was operationalized as children’s knowledge of the Secure Base Script with a coded narrative task. Children filled out questionnaires on supportive parenting, their environmental sensitivity and their depressive symptoms. Parents filled out questionnaires on children’s internalizing problems. Results: environmental sensitivity moderated the link between supportive parenting and attachment. More sensitive children that perceived their parents as less supportive less likely developed SBS knowledge. Further, environmental sensitivity moderated the link between SBS knowledge and the development of internalizing problems. More sensitive children with less SBS knowledge developed more internalizing problems. The findings support the importance of accounting for environmental sensitivity in attachment research.

Posttraumatic stress symptoms (PTS) have been observed in children exposed to family violence. Although functioning improves for many children after cessation of violence, pathways to recovery are poorly understood. This study tests the mediating pathways between changes in family violence and children’s PTS through children’s emotional security, parental stress, and parents’ PTS. We used longitudinal data of 562 children and their parents who were referred to child protection service. Data included three waves over a one and a half years period. Questionnaire data of both children and parents were analyzed in R Lavaan with Random Intercept Cross Lagged Panel Models to examine intrafamilial associations. Child-reported, but not parent-reported, decreases in family violence predicted decreases in child PTS from the first to the second wave. Changes in parental stress, parent PTS, and emotional security did not mediate the associations between change in family violence and child PTS. We found in exploratory analyses that decreases in parental stress predicted decreases in parent-reported family violence. The results emphasize the importance of reducing family violence for children to recover from PTS. Parental stress may be a factor in restoring safety.

Theoretical perspectives propose that positive childhood experiences (PCEs) are associated with adult mental health symptoms. The aim of the current study was to conduct a meta-analysis to evaluate the correlations between PCEs and adult mental health symptoms. 41 unique studies (N = 74,492) were included. Significant, negative, medium-to-large, effects were observed between PCEs and each mental health symptom (medium-to-large for overall mental health: r = −.268; and depression: r = −.273; for anxiety: r = −.246; and PTSD: r = −.243), indicating that higher levels of PCEs are linked to fewer mental health difficulties in adulthood. Meta-regression analyses identified current age at the time of mental health assessment and adverse childhood experiences (ACEs) as significant moderators. Specifically, the promotive effects of PCEs were stronger among younger adults and weakened with higher ACEs exposure, particularly in relation to overall adult mental health symptoms, depression, PTSD, and anxiety. In contrast, no significant moderation effects were found for sex or the type of PCEs measurement tool used. Integrated prevention frameworks that combine ACEs prevention with PCEs promotion can enhance mental health across the lifespan by addressing both risk and promotive pathways and providing developmentally tailored support.