Sitotroga cerealella (Olivier) (Lepidoptera: Gelechiidae) is one of the most serious pests of rice. The variations in phytoconstituents of rice cultivars (Miniket, Cottondora Sannalu, Gobindobhog, Swarna, and Tulaipanji) that led to differences in population growth of S. cerealella were studied. Fitness of the moth was approximated in terms of mean generation time (Tc), net reproductive rate (NRR or R0), intrinsic rate of population increase (rm), etc. Considering potential fecundity (Pf), growth rates, mortality coefficient (MC), and expected population size in the second generation (PF2), we classified the susceptibility of the rice cultivars to S. cerealella in the order as M > C > G > S > T. This was despite the higher concentrations of all the tested primary metabolites (PMs) in C and T. The least susceptibility of T and lower susceptibility of C than M can be attributed to their higher levels of secondary metabolites (SMs). M was highly susceptible due to its lowest SM content and sufficient levels of PMs. Now, G and S contained lower amounts of SMs, but they were moderately susceptible owing to the lower PM content. This emphasises the role of SMs, such as phenols, flavonoids, tannins, saponins, alkaloids, phytates, and oxalates, in determining the susceptibility of crop cultivars. Although none of the rice cultivars were entirely resistant, it can be stated that Miniket requires more protection measures against S. cerealella during storage than the others.

The crucifer flea beetle, Phyllotreta cruciferae (Goeze), and the striped flea beetle, P. striolata (Fabricius) (Coleoptera: Chrysomelidae), are invasive pests to canola, Brassica napus (Linnaeus) (Brassicaceae), in North America. Understanding how temperature and predators influence flea beetle damage in canola is critical for improving current management strategies, yet these factors are rarely studied. We examined the prevalence (proportion of plants damaged), intensity (defoliation of damaged plants), and overall damage (mean defoliation = prevalence × intensity) caused by flea beetles on canola seedlings at different temperatures, plant densities, and with or without generalist predators. Flea beetles preferred to feed on the abaxial (undersides) of cotyledons rather than the typically assessed adaxial (top) side, regardless of temperature, and produced more stem and cotyledon damage at higher temperatures, regardless of the species. Predators Pardosa spp. (Araneae: Lycosidae) and Pterostichus melanarius (Illiger) (Coleoptera: Carabidae) reduced flea beetle abundance and the intensity of cotyledon defoliation. Pterostichus melanarius also reduced overall stem damage, whereas Pardosa spp. reduced stem damage intensity. Under constant flea beetle densities, canola sustained less damage at a higher plant density. These results suggest that reducing the number of flea beetles per seedling, through predation or higher plant density, may help reduce Phyllotreta damage to canola.

Obesity is characterised by chronic low-grade inflammation, which is a key factor in the development of obesity-related co-morbidities. Intake of n-3 long-chain PUFAs is associated with anti-inflammatory effects. Recent studies suggest that also n-11 long-chain MUFAs may reduce the concentrations of inflammatory markers, possibly by increasing the biosynthesis of EPA. The primary aim was to investigate if diets added herring oil containing cetoleic acid (CA, C22:1n-11) or a CA concentrate (CECO) affected the fatty acid composition in tissues from obese rats with chronic inflammation. Secondary aims included investigating the effects on inflammatory markers. Thirty male obese Zucker fa/fa rats were fed diets containing herring oil (HERO) or a CECO, containing 0·70 or 1·40 wt% CA, respectively, with a comparable content of EPA (0·17 and 0·20 wt%, respectively), or a control diet with soyabean oil for 5 weeks. Data were analysed using one-way ANOVA. CA from HERO and CECO diets were recovered in liver, adipose tissue, muscle and blood cells. The EPA concentration was similar between HERO and CECO groups in tissues, whereas the hepatic concentrations of fatty acid desaturases were lower or similar to Controls. The concentrations of TNFα, matrix metalloproteinase-3, IL6, monocyte chemotactic protein 1 and integrin α M in adipose tissue, and the hepatic concentration of CD68 were lower after CECO intake but were not affected by the HERO diet. To conclude, rats fed the CECO diet had lower concentrations of inflammatory and macrophage infiltration markers, but this effect was probably not mediated through increased EPA biosynthesis.

There is growing evidence that optimising dietary quality and engaging in physical activity (PA) can reduce dementia and cognitive decline risk and improve psychosocial health and quality of life (QoL). Multimodal interventions focusing on diet and PA are recognised as significant strategies to tackle these behavioural risk factors; however, the cost-effectiveness of such interventions is seldom reported. A limited cost consequence based on a 12-month cluster-randomised Mediterranean diet (MedDiet) and walking controlled trial (MedWalk) was undertaken. In addition, QoL data were analysed. Programme costs ($AUD2024) covered staff to deliver the MedWalk programme and foods to support dietary behaviour change. The primary outcome measure of this study was change in QoL utility score, measured using the Assessment of Quality of Life (AQoL-8D). Change scores were compared for the groups using general linear models while controlling for demographic factors associated with baseline group differences and attrition. Change in QoL (decreased, maintained or improved) was determined using a cross-tabulation test. MedWalk programme costs were estimated at $2695 AUD per participant and control group cost at $165 per person – a differential cost of $2530. Mean change in utility scores from baseline to 12 months was not statistically significant between groups. Nevertheless, the MedWalk group was significantly less likely to experience a reduction in their QoL (20·3 % MedWalk v. 42·6 % control group) (P = 0·020). A MedDiet and walking intervention may have a role in preventing decline in QoL of older Australians; however, longer-term follow-up would be beneficial to see if this is maintained.

In the summer of 2016, a case of potentially herbicide-resistant redroot pigweed (Amaranthus retroflexus L.) was reported in Saint-Louis-de-Gonzague (QC, Canada) in an identity-preserved soybean [Glycine max (L.) Merr.] field where imazethapyr and chlorimuron-ethyl were applied preemergence, two active ingredients that inhibit acetolactate synthase (ALS). A few years earlier, several A. retroflexus populations resistant to ALS-inhibiting herbicides were reported in Manitoba (MB). Amaranthus retroflexus samples from these fields were collected and analyzed to characterize the presence of a resistance mechanism and the associated level of resistance. Sequencing of the ALS gene revealed a mutation resulting in a serine to asparagine substitution at amino acid position 653. Dose–response experiments indicated resistance factors of 27.7 to 194.0 to imazethapyr (imidazolinones) among these populations, but susceptibility to members of all other ALS inhibitor families that were tested. In one MB population, a serine to isoleucine substitution at position 653 was observed for the first time in A. retroflexus in some of the plants, and this contributed to the detection of resistance to thifensulfuron-methyl when compared with most susceptible control population. The response of the two MB control populations to thifensulfuron-methyl differed, and resistance factors were influenced by the level of susceptibility of the control populations to thifensulfuron-methyl. The same was not observed with imazethapyr. Non–target site resistance (NTSR) was not detected in the MB populations when they were exposed to malathion before exposure to the herbicide. We identified two new mutations on the ALS gene in A. retroflexus that result in ALS inhibitor–specific cross-resistance patterns. Further investigation into NTSR and the mechanism behind the differential response of the control populations to thifensulfuron-methyl are warranted.

The objective of this study was to evaluate the impact of incorporating group-based nutritional education into usual care on adherence to nutritional counselling among outpatients with type 2 diabetes (T2DM). This parallel-group, randomised controlled superiority trial was conducted with outpatients diagnosed with T2DM and poor glycaemic control. The control group received individual usual care, while the intervention group received usual care plus group-based nutritional education, which included three sessions: ‘Let’s Go Shopping,’ ‘Healthy Plate’ and ‘Hunger and Satiety.’ Baseline and four-month follow-up assessments were conducted, and outcome measures included adherence to nutritional counselling, glycaemic control, weight and physical activity. A total of 213 participants were included: 61·9 % were women, aged 60 years (interquartile range (IQR) = 53–65), 67·6 % with low socio-economic status, 67·3 % had obesity and the glycated Hb (HbA1c) was 9·3 % (IQR = 8·3–10·2 %). The intervention did not result in superior glycaemic control; however, short-term reductions in HbA1c were observed across all participants (–0·3 % (IQR = –0·7–0·3 %)). The intervention group demonstrated an increase in physical activity levels between visits (262 metabolic equivalent tasks (IQR = 99–257); P = 0·038), but no significant difference between the groups. Participants in the intervention group adhered to 67 % of combinations, compared with 50 % in the control group (P = 0·012), and adherence to the diabetes plate method was significantly higher in this group (75·9 % v. 62·2 %; P = 0·05). In conclusion, no significant between-group differences in glycaemic control were found, although both groups improved HbA1c short-term. Group-based nutritional education increased dietary adherence, particularly to the diabetes plate method and physical activity. Long-term effects on metabolic outcomes warrant further study.

Sepsis affects 50 million people globally, contributing to 20 % of all deaths and significantly increasing healthcare costs due to intensive care needs. Although the role of n-3 fatty acids in reducing sepsis mortality remains debated, recent studies suggest their potential in modulating immune responses and improving outcomes. This umbrella review aims to clarify the benefits of n-3 supplementation on mortality rate, length of intensive care unit (ICU) stays and days on mechanical ventilation in patients with sepsis. Following Cochrane and Grading of Recommendations Assessment, Development, and Evaluation (GRADE) methodologies, a systematic search was conducted across multiple databases up to February 2025. After independent screening, data extraction and critical appraisal, meta-analyses were reassessed using the DerSimonian and Laird model. Evidence was graded using the GRADE approach, categorising outcomes based on strength and quality. A comprehensive search identified 934 records, of which thirty-four randomised controlled trials (RCT) from twenty-one systematic reviews and meta-analyses focused on n-3 supplementation in sepsis patients. n-3 significantly reduced mortality (risk ratio: 0·79, 95 % CI 0·69, 0·90), length of ICU stays (mean difference (MD): −3·6 d, 95 % CI −4·39, −2·81) and ventilation days (MD: −2·86 d, 95 % CI −4·46, −1·26). Parenteral nutrition showed slightly better outcomes than enteral nutrition, and EPA and DHA provided superior results compared with mixed oils. These findings suggest n-3 supplementation could improve mortality, ICU stays and ventilator dependency in patients with sepsis. However, the certainty of the evidence ranges from low to very low, emphasising the need for further high-quality RCT to validate these benefits. Also, clinicians should prescribe n-3 supplements cautiously in this regard.

The right to roam – balancing inclusion and enclosure. In Norway, the right to roam is an old custom – a right to traverse and gather berries, herbs and firewood on uncultivated lands – dating back to the Viking Age. In 1957, this right was included in Norwegian laws, in the Outdoor Life Act (Friluftslova). The law transformed agrarian lands into areas for outdoor life and recreation, primarily walking and hiking. However, due to modernisation, the activities performed today are very different than those in the 1950s, involving many sorts of technical devices and installations, commercial activities and a different landscape. The law was a manifestation of the Norwegian outfields as a commons, but what is a commons for some can be an enclosure for others. This is the topic of this article: how the right to roam includes many and much but represents encroachment, displacements and enclosures and has created crowding, natural wear and tear and urges for management regimes. The article describes this as a balance between inclusion and enclosure. The article has two major parts: one presenting the academic discussion about inclusion and enclosure, the other discussing the implementation of the principle in Norway in light of this theory.