Using Patterson–Sullivan measures, we investigate growth problems for groups acting on a metric space with a strongly contracting element.

This paper is concerned with stochastic Schrödinger delay lattice systems with both locally Lipschitz drift and diffusion terms. Based on the uniform estimates and the equicontinuity of the segment of the solution in probability, we show the tightness of a family of probability distributions of the solution and its segment process, and hence the existence of invariant measures on $l^2\times L^2((-\rho,\,0);l^2)$ with $\rho >0$. We also establish a large deviation principle for the solutions with small noise by the weak convergence method.

In this paper, we analyse Turing instability and bifurcations in a host–parasitoid model with nonlocal effect. For a ordinary differential equation model, we provide some preliminary analysis on Hopf bifurcation. For a reaction–diffusion model with local intraspecific prey competition, we first explore the Turing instability of spatially homogeneous steady states. Next, we show that the model can undergo Hopf bifurcation and Turing–Hopf bifurcation, and find that a pair of spatially nonhomogeneous periodic solutions is stable for a (8,0)-mode Turing–Hopf bifurcation and unstable for a (3,0)-mode Turing–Hopf bifurcation. For a reaction–diffusion model with nonlocal intraspecific prey competition, we study the existence of the Hopf bifurcation, double-Hopf bifurcation, Turing bifurcation, and Turing–Hopf bifurcation successively, and find that a spatially nonhomogeneous quasi-periodic solution is unstable for a (0,1)-mode double-Hopf bifurcation. Our results indicate that the model exhibits complex pattern formations, including transient states, monostability, bistability, and tristability. Finally, numerical simulations are provided to illustrate complex dynamics and verify our theoretical results.

We generalise and improve some recent bounds for additive energies of modular roots. Our arguments use a variety of techniques, including those from additive combinatorics, algebraic number theory and the geometry of numbers. We give applications of these results to new bounds on correlations between Salié sums and to a new equidistribution estimate for the set of modular roots of primes.

Let G be a countable residually finite group (for instance, ${\mathbb F}_2$) and let $\overleftarrow {G}$ be a totally disconnected metric compactification of G equipped with the action of G by left multiplication. For every $r\geq 1$, we construct a Toeplitz G-subshift $(X,\sigma ,G)$, which is an almost one-to-one extension of $\overleftarrow {G}$, having r ergodic measures $\nu _1, \ldots ,\nu _r$ such that for every $1\leq i\leq r$, the measure-theoretic dynamical system $(X,\sigma ,G,\nu _i)$ is isomorphic to $\overleftarrow {G}$ endowed with the Haar measure. The construction we propose is general (for amenable and non-amenable residually finite groups); however, we point out the differences and obstructions that could appear when the acting group is not amenable.

In this paper, we study the existence of solutions for a critical time–harmonic Maxwell equation in nonlocal media

\[ \begin{cases} \nabla\times(\nabla\times u)+\lambda u=\left(I_{\alpha}\ast|u|^{2^{{\ast}}_{\alpha}}\right)|u|^{2^{{\ast}}_{\alpha}-2}u & \mathrm{in}\ \Omega,\\ \nu\times u=0 & \mathrm{on}\ \partial\Omega, \end{cases} \]

$\Omega \subset \mathbb {R}^{3}$

$\mathcal {C}^{1,1}$

$\nu$

$\lambda <0$

$2^{\ast }_{\alpha }=3+\alpha$

$0<\alpha <3$

$W^{\alpha,2^{\ast }_{\alpha }}_{0}(\mathrm {curl};\Omega )$

A set of complex numbers $S$ is called invariant if it is closed under addition and multiplication, namely, for any $x, y \in S$ we have $x+y \in S$ and $xy \in S$. For each $s \in {\mathbb {C}}$ the smallest invariant set ${\mathbb {N}}[s]$ containing $s$ consists of all possible sums $\sum _{i \in I} a_i s^i$, where $I$ runs over all finite nonempty subsets of the set of positive integers ${\mathbb {N}}$ and $a_i \in {\mathbb {N}}$ for each $i \in I$. In this paper, we prove that for $s \in {\mathbb {C}}$ the set ${\mathbb {N}}[s]$ is everywhere dense in ${\mathbb {C}}$ if and only if $s \notin {\mathbb {R}}$ and $s$ is not a quadratic algebraic integer. More precisely, we show that if $s \in {\mathbb {C}} \setminus {\mathbb {R}}$ is a transcendental number, then there is a positive integer $n$ such that the sumset ${\mathbb {N}} t^n+{\mathbb {N}} t^{2n} +{\mathbb {N}} t^{3n}$ is everywhere dense in ${\mathbb {C}}$ for either $t=s$ or $t=s+s^2$. Similarly, if $s \in {\mathbb {C}} \setminus {\mathbb {R}}$ is an algebraic number of degree $d \ne 2, 4$, then there are positive integers $n, m$ such that the sumset ${\mathbb {N}} t^n+{\mathbb {N}} t^{2n} +{\mathbb {N}} t^{3n}$ is everywhere dense in ${\mathbb {C}}$ for $t=ms+s^2$. For quadratic and some special quartic algebraic numbers $s$ it is shown that a similar sumset of three sets cannot be dense. In each of these two cases the density of ${\mathbb {N}}[s]$ in ${\mathbb {C}}$ is established by a different method: for those special quartic numbers, it is possible to take a sumset of four sets.

The complete classification of the finite simple groups that are $(2,3)$-generated is a problem which is still open only for orthogonal groups. Here, we construct $(2, 3)$-generators for the finite odd-dimensional orthogonal groups $\Omega _{2k+1}(q)$, $k\geq 4$. As a byproduct, we also obtain $(2,3)$-generators for $\Omega _{4k}^+(q)$ with $k\geq 3$ and q odd, and for $\Omega _{4k+2}^\pm (q)$ with $k\geq 4$ and $q\equiv \pm 1~ \mathrm {(mod~ 4)}$.

We formulate haptotaxis models of cancer invasion wherein the infiltrating cancer cells can occupy a spectrum of states in phenotype space, ranging from ‘fully mesenchymal’ to ‘fully epithelial’. The more mesenchymal cells are those that display stronger haptotaxis responses and have greater capacity to modify the extracellular matrix (ECM) through enhanced secretion of matrix-degrading enzymes (MDEs). However, as a trade-off, they have lower proliferative capacity than the more epithelial cells. The framework is multiscale in that we start with an individual-based model that tracks the dynamics of single cells, which is based on a branching random walk over a lattice representing both physical and phenotype space. We formally derive the corresponding continuum model, which takes the form of a coupled system comprising a partial integro-differential equation for the local cell population density function, a partial differential equation for the MDE concentration and an infinite-dimensional ordinary differential equation for the ECM density. Despite the intricacy of the model, we show, through formal asymptotic techniques, that for certain parameter regimes it is possible to carry out a detailed travelling wave analysis and obtain invading fronts with spatial structuring of phenotypes. Precisely, the most mesenchymal cells dominate the leading edge of the invasion wave and the most epithelial (and most proliferative) dominate the rear, representing a bulk tumour population. As such, the model recapitulates similar observations into a front to back structuring of invasion waves into leader-type and follower-type cells, witnessed in an increasing number of experimental studies over recent years.

The paper is concerned with positive solutions to problems of the type

\[ -\Delta_{\mathbb{B}^{N}} u - \lambda u = a(x) |u|^{p-1}\;u + f \text{ in }\mathbb{B}^{N}, \quad u \in H^{1}{(\mathbb{B}^{N})}, \]

$\mathbb {B}^N$

$1< p<2^*-1:=\frac {N+2}{N-2}$

$\;\lambda < \frac {(N-1)^2}{4}$

$f \in H^{-1}(\mathbb {B}^{N})$

$f \not \equiv 0$

$a\in L^\infty (\mathbb {B}^N)$

$\lim _{d(x, 0) \rightarrow \infty } a(x) \rightarrow 1,$

$d(x,\, 0)$

$a(x) \leq 1$

$a(x) \geq 1$

$\mu ( \{ x : a(x) \neq 1\}) > 0.$

$a(x) \equiv 1$

In this paper, we consider random iterations of polynomial maps $z^{2} + c_{n}$, where $c_{n}$ are complex-valued independent random variables following the uniform distribution on the closed disk with center c and radius r. The aim of this paper is twofold. First, we study the (dis)connectedness of random Julia sets. Here, we reveal the relationships between the bifurcation radius and connectedness of random Julia sets. Second, we investigate the bifurcation of our random iterations and give quantitative estimates of bifurcation parameters. In particular, we prove that for the central parameter $c = -1$, almost every random Julia set is totally disconnected with much smaller radial parameters r than expected. We also introduce several open questions worth discussing.

We generalize the known collision results for a solid in a 3D compressible Newtonian fluid to compressible non-Newtonian ones, and to Newtonian fluids with temperature-depending viscosities.

In this paper, we consider the following non-linear system involving the fractional Laplacian0.1

\begin{equation} \left\{\begin{array}{@{}ll} (-\Delta)^{s} u (x)= f(u,\,v), \\ (-\Delta)^{s} v (x)= g(u,\,v), \end{array} \right. \end{equation}

$0< s<1$

$x_n$

In this note, we prove a formula for the cancellation exponent $k_{v,n}$ between division polynomials $\psi _n$ and $\phi _n$ associated with a sequence $\{nP\}_{n\in \mathbb {N}}$ of points on an elliptic curve $E$ defined over a discrete valuation field $K$. The formula greatly generalizes the previously known special cases and treats also the case of non-standard Kodaira types for non-perfect residue fields.